Mitochondrial Dysfunction in Alzheimer’s Disease and the Rationale for Bioenergetics Based Therapies

Cites (205)

Items 201–205 of 205

1. Age-associated changes in basal NF-κB function in human CD4+ T lymphocytes via dysregulation of PI3 kinase
   Arsun Bektas, Yongqing Zhang, Elin Lehmann, William H. Wood, Kevin G. Becker, Karen Madara, Luigi Ferrucci, Ranjan Sen · 2014 · Aging
2. Mutations in mitochondrial‐encoded cytochrome c oxidase subunits I, II, and III genes detected in Alzheimer's disease using single‐strand conformation polymorphism
   Natasha S. Hamblet, Brian D. Ragland, Mervat Ahmed Ali, Barbara Conyers, Frank J. Castora · 2005 · Electrophoresis
3. Insulin-stimulated phosphorylation of endothelial nitric oxide synthase at serine-615 contributes to nitric oxide synthesis
   Stuart A. Ritchie, Christine F. Kohlhaas, Alasdair R. Boyd, Krishna C. Yalla, Kenneth Walsh, John Connell, Ian P. Salt · 2009 · Biochemical Journal
4. FDG and Amyloid PET in Cognitively Normal Individuals at Risk for Late-Onset Alzheimer’s Disease
   John Murray, Wai Hon Tsui, Yi Li, Pauline F. McHugh, Schantel Williams, Megan Cummings, Elizabeth Pirraglia, Lilja B. Sólnes, Ricardo S. Osorio, Lidia Glodzik et al. · 2014 · Advances in Molecular Imaging
5. Abnormal nitric oxide production in aged rat mesenteric arteries is mediated by NAD(P)H oxidase-derived peroxide
   Xiaosun Zhou, Harold Glenn Bohlen, Joseph L. Unthank, Steven J. Miller · 2009 · American Journal of Physiology-Heart and Circulatory Physiology