Identification of factor H-like protein 1 as the predominant complement regulator in Bruch’s membrane: Implications for age-related macular degeneration

Clark, Simon J, Schmidt, Christoph Q, White, Anne M, Hakobyan, Svetlana, Morgan, B Paul, Bishop, Paul N·2014·The Journal of Immunology

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Items 101–122 of 122

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  3. Enrichment of Bruch's Membrane from Human Donor Eyes
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  4. Complement Factor H Gene Mutations: Implications for Genetic Testing and Precision Medicine in Macular Degeneration
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  7. Shifting From Systemic to Precision‐Targeted Complement Therapies: Opportunities and Hurdles
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  10. Modeling Macular Degeneration Using Quantitative Phenotypes
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  11. Faculty Opinions recommendation of A genome-wide association study identifies key modulators of complement factor H binding to malondialdehyde-epitopes.
    Toshiyuki Miyata · 2020 · Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature
  12. FH-like protein 1-Y402H mutation promoted microglial synapse phagocytosis and cognitive impairment in Alzheimer's disease model
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  13. Preclinical development of a dual targeting bicistronic gene therapy approach for the treatment of wet age-related macular degeneration
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  22. Diagnostic interpretation of CFH expression in pan-cancer analyses: the importance of factor H–like 1 isoform resolution
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