Clinical Disorders of Bone and Mineral Metabolism

Abstract

Symposium1 November 1983Clinical Disorders of Bone and Mineral MetabolismBOY FRAME, M.D., GEOFFREY M. MAREL, M.D.BOY FRAME, M.D.Search for more papers by this author, GEOFFREY M. MAREL, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-99-5-725 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptApproximately 600 physicians and medical scientists from more than 30 countries attended the Frances and Anthony D'Anna International Memorial Symposium on Clinical Disorders of Bone and Mineral Metabolism held on 8 to 13 May 1983 in Dearborn, Michigan. Topics discussed at the symposium included clinical applications of parathyroid hormone assays; assessment of trabecular bone status; the role of cholecalciferol in clinical medicine; an assessment of mineral and matrix turnover; pathogenesis of hyperparathyroidism; management of hypercalcemic hyperparathyroidism; recent developments in bone physiology; secondary hyperparathyroidism; parathyroid-hormone-mediated bone loss; osteomalacia and nutritional bone disease; renal osteodystrophy; nonparathyroid hypercalcemia; osteoporosis; Paget's disease; idiopathic hypercalciuria;...Symposium Papers1. SEGRE G, HARRIS S, MAGLIARO D, TULLY G, NEER R, and POTTS J. Amino-terminal radioimmunoassay for human parathyroid hormone. Massachusetts General Hospital; Boston, Massachusetts. Google Scholar2. GOLTZMAN D, GOMOLIN H, WEXLER M, and MEAKINS J. Analysis of parathyroid hormone-like substances in hyperparathyroid states. Royal Victoria Hospital and McGill University; Montreal, Canada. Google Scholar3. TALPOS G, KLEEREKOPER M, KAMBOURIS A, and MOORE D. Management of primary hyperparathyroidism: report on a workshop discussing a proposed clinical study to prospectively evaluate the benefits and risks of surgical and non-surgical approaches to mild hyperparathyroidism. Henry Ford Hospital; Detroit, Michigan. Google Scholar4. POSEN S, CLIFTON-BLIGH P, REEVE T, WAGSTAFFE C, and WILKINSON M. Is parathyroidectomy of benefit in primary hyperparathyroidism? Royal North Shore Hospital; St. Leonards, New South Wales, Australia. Google Scholar5. MARCUS R, MADVIG P, CRIM M, KOSEK J, and PONT A. Estrogen therapy for postmenopausal women with primary hyperparathyroidism. Veterans Administration Medical Center and Stanford University; Palo Alto, California. Google Scholar6. VAN DOP C and BOURNE H. Molecular basis of pseudohypoparathyroidism. University of California; San Francisco, California. Google Scholar7. LEVINE M and SPIEGEL A. Multiple hormone resistance in pseudohypoparathyroidism. Johns Hopkins University School of Medicine; Baltimore, Maryland. Google Scholar8. HAUSSLER M. Vitamin D metabolites: assay methodology, clinical application and new physiologic insights. University of Arizona Health Sciences Center; Tucson, Arizona. Google Scholar9. DELUCA H. The cardinal role of 1,25-dihydroxyvitamin D3 in mineral homeostasis. University of Wisconsin; Madison, Wisconsin. Google Scholar10. RASMUSSEN H. The role of 1,25(OH)2D3 in the pathogenesis of osteomalacia. Yale University; New Haven, Connecticut. Google Scholar11. RAO D, VILLANUEVA A, MATHEWS M, PUMO B, FRAME B, and PARFITT M. Histologic evolution of vitamin-D depletion (D—). Henry Ford Hospital; Detroit, Michigan. Google Scholar12. OTT S, MALONEY N, and KLEIN G. Aluminum is associated with low bone formation rate in patients on chronic parenteral nutrition. University of Washington; Seattle, Washington. Google Scholar13. ACKRILL P and DAY J. The management of aluminum related osteomalacia in renal failure. Withington Hospital and Manchester University; Manchester, United Kingdom. Google Scholar14. BROADUS A and STEWART A. Humoral hypercalcemia of malignancy. Yale University School of Medicine: New Haven, Connecticut. Google Scholar15. KRONENBERG H. Possible use of recombinant DNA technology to characterize humoral hypercalcemia factors. Massachusetts General Hospital; Boston, Massachusetts. Google Scholar16. PARFITT A. Assessment of trabecular bone status. Henry Ford Hospital; Detroit, Michigan. Google Scholar17. MAZESS R. Problems of trabecular bone measurement. University of Wisconsin; Madison, Wisconsin. Google Scholar18. GENANT H, CANN C, BOYD D, RUTT B, and STEBLER B. Quantitative computed tomography for vertebral mineral determination: I. Technical. University of California; San Francisco, California. Google Scholar19. PARFITT A, MATHEWS C, VILLANUEVA A, RAO D, ROGERS M, and KLEEREKOPER M. Microstructural and cellular basis of age related bone loss (ARBL) and osteoporosis (OP). Henry Ford Hospital; Detroit, Michigan. Google Scholar20. HEANEY R. Determinants of age-related bone loss. Creighton University; Omaha, Nebraska. Google Scholar21. RIGGS B, MELTON L, and WAHNER H. Heterogeneity of involutional osteoporosis. Mayo Clinic and Foundation; Rochester, Minnesota. Google Scholar22. LINDSA