Crossley, Merlin

FOG, a Multitype Zinc Finger Protein, Acts as a Cofactor for Transcription Factor GATA-1 in Erythroid and Megakaryocytic Differentiation

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Transcription factors LRF and BCL11A independently repress expression of fetal hemoglobin

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Natural regulatory mutations elevate the fetal globin gene via disruption of BCL11A or ZBTB7A binding

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Isolation and characterization of the cDNA encoding BKLF/TEF-2, a major CACCC-box-binding protein in erythroid cells and selected other cells

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Self-Association of the Erythroid Transcription Factor GATA-1 Mediated by Its Zinc Finger Domains

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The C-Terminal Zinc Finger of GATA-1 or GATA-2 Is Sufficient To Induce Megakaryocytic Differentiation of an Early Myeloid Cell Line

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Protein-Protein Interaction between Fli-1 and GATA-1 Mediates Synergistic Expression of Megakaryocyte-Specific Genes through Cooperative DNA Binding

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Wake-up Sleepy Gene: Reactivating Fetal Globin for β-Hemoglobinopathies

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Protein interactions: is seeing believing?

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Multi-species, multi-transcription factor binding highlights conserved control of tissue-specific biological pathways

Ballester, B., Medina-Rivera, A., Schmidt, D., Gonzàlez-Porta, M., Carlucci, M., Chen, X., Chessman, K., Faure, A. J., Funnell, A. P., Goncalves, A., et al. (2014) · eLife

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PU.1 and Haematopoietic Cell Fate: Dosage Matters

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KLF1 directly activates expression of the novel fetal globin repressor ZBTB7A/LRF in erythroid cells

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Molecular Analysis of the Interaction between the Hematopoietic Master Transcription Factors GATA-1 and PU.1

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