Finch, Andrew J.
Queen Mary University of London · Edinburgh Cancer Research · Institute of Genetics and Cancer · Barts Cancer Institute · University of Edinburgh
5 papers on CoChatORCID 0000-0002-8065-4623
Most cited papers
- Verified
The pathological response to DNA damage does not contribute to p53-mediated tumour suppression
Christophorou, M. A., Ringshausen, I., Finch, A. J., Swigart, L. B., & Evan, G. I. (2006) · Nature
Cancer-related Molecular PathwaysDNA Repair Mechanisms
Cited by 382
- Verified
Mdm2 is critically and continuously required to suppress lethal p53 activity in vivo
Ringshausen, I., O'Shea, C. C., Finch, A. J., Swigart, L. B., & Evan, G. I. (2006) · Cancer Cell
Cancer-related Molecular PathwaysCell death mechanisms and regulation
Cited by 256
- Verified
The innate immune sensor Toll-like receptor 2 controls the senescence-associated secretory phenotype
Hari, P., Millar, F. R., Tarrats, N., Birch, J., Quintanilla, A., Rink, C. J., Fernández-Duran, I., Muir, M., Finch, A. J., Brunton, V. G., et al. (2019) · Science Advances
Telomeres, Telomerase, and SenescenceImmune Response and Inflammation
Cited by 154
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Fumarate hydratase is a critical metabolic regulator of hematopoietic stem cell functions
Guitart, A. V., Panagopoulou, T. I., Villacreces, A., Vukovic, M., Sepulveda, C., Allen, L., Carter, R. N., van de Lagemaat, L. N., Morgan, M., Giles, P., et al. (2017) · Journal of Experimental Medicine
Hematopoietic Stem Cell TransplantationCancer, Hypoxia, and Metabolism
Cited by 86
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Reversal of Mitochondrial Malate Dehydrogenase 2 Enables Anaplerosis via Redox Rescue in Respiration-Deficient Cells
Altea-Manzano, P., Vandekeere, A., Edwards-Hicks, J., Roldan, M., Abraham, E., Lleshi, X., Guerrieri, A. N., Berardi, D., Wills, J., Junior, J. M., et al. (2022) · Molecular Cell
Mitochondrial Function and PathologyCancer, Hypoxia, and Metabolism
Cited by 57
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