Cramer, Thorsten

HIF-1α is essential for myeloid cell-mediated inflammation

Cramer, T., Yamanishi, Y., Clausen, B. E., Förster, I., Pawlinski, R., Mackman, N., Haase, V. H., Jaenisch, R., Corr, M., Nizet, V., et al. (2003) · Cell

Cancer, Hypoxia, and MetabolismImmune cells in cancer

Cited by 2,046

The return of metabolism: biochemistry and physiology of the pentose phosphate pathway

Stincone, A., Prigione, A., Cramer, T., Wamelink, M. M. C., Campbell, K., Cheung, E., Olin‐Sandoval, V., Grüning, N., Krüger, A., Tauqeer Alam, M., et al. (2015) · Biological Reviews

Biochemical and Molecular ResearchRedox biology and oxidative stress

Cited by 1,576

Hypoxia-induced neutrophil survival is mediated by HIF-1α–dependent NF-κB activity

Walmsley, S. R., Print, C., Farahi, N., Peyssonnaux, C., Johnson, R. S., Cramer, T., Sobolewski, A., Condliffe, A. M., Cowburn, A. S., Johnson, N., et al. (2005) · The Journal of Experimental Medicine

Cancer, Hypoxia, and MetabolismNeutrophil, Myeloperoxidase and Oxidative Mechanisms

Cited by 891

A microfluidics platform for combinatorial drug screening on cancer biopsies

Eduati, F., Utharala, R., Madhavan, D., Neumann, U. P., Longerich, T., Cramer, T., Saez-Rodriguez, J., & Merten, C. A. (2018) · Nature Communications

Microfluidic and Capillary Electrophoresis ApplicationsInnovative Microfluidic and Catalytic Techniques Innovation

Cited by 239

The tumour microenvironment and immune milieu of cholangiocarcinoma

Fabris, L., Perugorria, M. J., Mertens, J., Björkström, N. K., Cramer, T., Lleo, A., Solinas, A., Sänger, H., Lukacs‐Kornek, V., Moncsek, A., et al. (2019) · Liver International

Cholangiocarcinoma and Gallbladder Cancer StudiesLiver Diseases and Immunity

Cited by 187

Glutamine deprivation counteracts hypoxia-induced chemoresistance

Wappler, J., Arts, M., Röth, A., Heeren, R. M. A., Peter Neumann, U., Olde Damink, S. W., Soons, Z., & Cramer, T. (2020) · Neoplasia

Cancer, Hypoxia, and MetabolismCholangiocarcinoma and Gallbladder Cancer Studies

Cited by 34

Gastrin transactivates the chromogranin A gene through MEK-1/ERK- and PKC-dependent phosphorylation of Sp1 and CREB.

Cramer, T., Jüttner, S., Plath, T., Mergler, S., Seufferlein, T., Wang, T. C., Merchant, J., & Höcker, M. (2008) · Cellular Signalling

Protein Kinase Regulation and GTPase SignalingReceptor Mechanisms and Signaling

Cited by 27