Hemmerich, Peter

Conserved senescence associated genes and pathways in primary human fibroblasts detected by RNA-seq

Marthandan, S., Baumgart, M., Priebe, S., Groth, M., Schaer, J., Kaether, C., Guthke, R., Cellerino, A., Platzer, M., Diekmann, S., et al. (2016) · PLOS ONE

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 91

Similarities in Gene Expression Profiles during In Vitro Aging of Primary Human Embryonic Lung and Foreskin Fibroblasts

Marthandan, S., Priebe, S., Baumgart, M., Groth, M., Cellerino, A., Guthke, R., Hemmerich, P., & Diekmann, S. (2015) · BioMed Research International

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 47

Conserved genes and pathways in primary human fibroblast strains undergoing replicative and radiation induced senescence

Marthandan, S., Menzel, U., Priebe, S., Groth, M., Guthke, R., Platzer, M., Hemmerich, P., Kaether, C., & Diekmann, S. (2016) · Biological Research

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 39

Quantitative model of cell cycle arrest and cellular senescence in primary human fibroblasts

Schäuble, S., Klement, K., Marthandan, S., Münch, S., Heiland, I., Schuster, S., Hemmerich, P., & Diekmann, S. (2012) · PLoS ONE

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 35

The Tumor Suppressor PML Specifically Accumulates at RPA/Rad51-Containing DNA Damage Repair Foci but Is Nonessential for DNA Damage-Induced Fibroblast Senescence

Münch, S., Weidtkamp-Peters, S., Klement, K., Grigaravicius, P., Monajembashi, S., Salomoni, P., Pandolfi, P. P., Weißhart, K., & Hemmerich, P. (2014) · Molecular and Cellular Biology

DNA Repair MechanismsTelomeres, Telomerase, and Senescence

Cited by 32