Han, Dong-Ho

High-fat diets cause insulin resistance despite an increase in muscle mitochondria

Hancock, C. R., Han, D.-H., Chen, M., Terada, S., Yasuda, T., Wright, D. C., & Holloszy, J. O. (2008) · Proceedings of the National Academy of Sciences

Adipose Tissue and MetabolismPeroxisome Proliferator-Activated Receptors

Cited by 538

Exercise-induced mitochondrial biogenesis begins before the increase in muscle PGC-1α expression.

Wright, D. C., Han, D.-H., Garcia-Roves, P. M., Geiger, P. C., Jones, T. E., & Holloszy, J. O. (2007) · Journal of Biological Chemistry

Adipose Tissue and MetabolismMitochondrial Function and Pathology

Cited by 488

Calcium Induces Increases in Peroxisome Proliferator-activated Receptor γ Coactivator-1α and Mitochondrial Biogenesis by a Pathway Leading to p38 Mitogen-activated Protein Kinase Activation

Wright, D. C., Geiger, P. C., Han, D.-H., Jones, T. E., & Holloszy, J. O. (2007) · Journal of Biological Chemistry

Adipose Tissue and MetabolismMetabolism, Diabetes, and Cancer

Cited by 309

Raising plasma fatty acid concentration induces increased biogenesis of mitochondria in skeletal muscle

Garcia-Roves, P., Huss, J. M., Han, D.-H., Hancock, C. R., Iglesias-Gutierrez, E., Chen, M., & Holloszy, J. O. (2007) · Proceedings of the National Academy of Sciences

Adipose Tissue and MetabolismPeroxisome Proliferator-Activated Receptors

Cited by 246

Activation of AMP kinase enhances sensitivity of muscle glucose transport to insulin

Fisher, J. S., Gao, J., Han, D.-H., Holloszy, J. O., & Nolte, L. A. (2002) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerAdipose Tissue and Metabolism

Cited by 244

The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2

Gan, Z., Burkart-Hartman, E. M., Han, D.-H., Finck, B., Leone, T. C., Smith, E. Y., Ayala, J. E., Holloszy, J., & Kelly, D. P. (2011) · Genes & Development

Peroxisome Proliferator-Activated ReceptorsMetabolism, Diabetes, and Cancer

Cited by 138

Does calorie restriction induce mitochondrial biogenesis? A reevaluation

Hancock, C. R., Han, D., Higashida, K., Kim, S. H., & Holloszy, J. O. (2011) · The FASEB Journal

Mitochondrial Function and PathologyGenetics, Aging, and Longevity in Model Organisms

Cited by 131

Prevention of glycogen supercompensation prolongs the increase in muscle GLUT4 after exercise

Garcia-Roves, P. M., Han, D.-H., Song, Z., Jones, T. E., Hucker, K. A., & Holloszy, J. O. (2003) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerMuscle metabolism and nutrition

Cited by 93

Calpain System Regulates Muscle Mass and Glucose Transporter GLUT4 Turnover

Otani, K., Han, D.-H., Ford, E. L., Garcia-Roves, P. M., Ye, H., Horikawa, Y., Bell, G. I., Holloszy, J. O., & Polonsky, K. S. (2004) · Journal of Biological Chemistry

Calpain Protease Function and RegulationMetabolism, Diabetes, and Cancer

Cited by 67

UCP-mediated energy depletion in skeletal muscle increases glucose transport despite lipid accumulation and mitochondrial dysfunction

Han, D.-H., Nolte, L. A., Ju, J.-S., Coleman, T., Holloszy, J. O., & Semenkovich, C. F. (2004) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerAdipose Tissue and Metabolism

Cited by 49

IL-6 increases muscle insulin sensitivity only at superphysiological levels

Geiger, P. C., Hancock, C., Wright, D. C., Han, D.-H., & Holloszy, J. O. (2007) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerExercise and Physiological Responses

Cited by 36

Inhibition of calpain results in impaired contraction-stimulated GLUT4 translocation in skeletal muscle

Otani, K., Polonsky, K. S., Holloszy, J. O., & Han, D.-H. (2006) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerCalpain Protease Function and Regulation

Cited by 14