Schulman, Howard

LTP induction by structural rather than enzymatic functions of CaMKII

Tullis, J. E., Larsen, M. E., Rumian, N. L., Freund, R. K., Boxer, E. E., Brown, C. N., Coultrap, S. J., Schulman, H., Aoto, J., Dell’Acqua, M. L., et al. (2023) · Nature

Neuroscience and Neuropharmacology ResearchMemory and Neural Mechanisms

Cited by 112

Flexible linkers in CaMKII control the balance between activating and inhibitory autophosphorylation

Bhattacharyya, M., Lee, Y. K., Muratcioglu, S., Qiu, B., Nyayapati, P., Schulman, H., Groves, J. T., & Kuriyan, J. (2020) · eLife

Protein Kinase Regulation and GTPase SignalingNeuroscience and Neuropharmacology Research

Cited by 58

The intellectual disability-associated CAMK2G p.Arg292Pro mutation acts as a pathogenic gain-of-function

Proietti Onori, M., Koopal, B., Everman, D. B., Worthington, J. D., Jones, J. R., Ploeg, M. A., Mientjes, E., van Bon, B. W., Kleefstra, T., Schulman, H., et al. (2018) · Human Mutation

Genetics and Neurodevelopmental DisordersGenomics and Rare Diseases

Cited by 39