Fujita, Kyota
Tokyo Medical and Dental University
5 papers on CoChatORCID 0000-0001-8384-8143
Most cited papers
- Verified
CD38 is critical for social behaviour by regulating oxytocin secretion
Jin, D., Liu, H.-X., Hirai, H., Torashima, T., Nagai, T., Lopatina, O., Shnayder, N. A., Yamada, K., Noda, M., Seike, T., et al. (2007) · Nature
Neuroendocrine regulation and behaviorNeuropeptides and Animal Physiology
Cited by 665
- Verified
Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation
Jin, M., Shiwaku, H., Tanaka, H., Obita, T., Ohuchi, S., Yoshioka, Y., Jin, X., Kondo, K., Fujita, K., Homma, H., et al. (2021) · Nature Communications
Neuroinflammation and Neurodegeneration MechanismsAlzheimer's disease research and treatments
Cited by 223
- Verified
HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer’s disease
Fujita, K., Motoki, K., Tagawa, K., Chen, X., Hama, H., Nakajima, K., Homma, H., Tamura, T., Watanabe, H., Katsuno, M., et al. (2016) · Scientific Reports
Alzheimer's disease research and treatmentsAdvanced Glycation End Products research
Cited by 160
- Verified
YAP-dependent necrosis occurs in early stages of Alzheimer’s disease and regulates mouse model pathology
Tanaka, H., Homma, H., Fujita, K., Kondo, K., Yamada, S., Jin, X., Waragai, M., Ohtomo, G., Iwata, A., Tagawa, K., et al. (2020) · Nature Communications
Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research
Cited by 119
- Verified
Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimer's disease brain
Tagawa, K., Homma, H., Saito, A., Fujita, K., Chen, X., Imoto, S., Oka, T., Ito, H., Motoki, K., Yoshida, C., et al. (2015) · Human Molecular Genetics
Alzheimer's disease research and treatmentsAdvanced Proteomics Techniques and Applications
Cited by 86
Go further with a free account
Verify every reference, keep papers in a library, and ask questions across them.
Sign up - it is free