Oishi, Hisashi

Restoring oxidant signaling suppresses proarthritogenic T cell effector functions in rheumatoid arthritis

Yang, Z., Shen, Y., Oishi, H., Matteson, E. L., Tian, L., Goronzy, J. J., & Weyand, C. M. (2016) · Science Translational Medicine

Rheumatoid Arthritis Research and TherapiesRedox biology and oxidative stress

Cited by 275

Deficiency of the DNA repair enzyme ATM in rheumatoid arthritis.

Shao, L., Fujii, H., Colmegna, I., Oishi, H., Goronzy, J. J., & Weyand, C. M. (2009) · Journal of Experimental Medicine

DNA Repair MechanismsRheumatoid Arthritis Research and Therapies

Cited by 159

p62/SQSTM1 and Nrf2 are essential for exercise‐mediated enhancement of antioxidant protein expression in oxidative muscle

Yamada, M., Iwata, M., Warabi, E., Oishi, H., Lira, V. A., & Okutsu, M. (2019) · The FASEB Journal

Genomics, phytochemicals, and oxidative stressExercise and Physiological Responses

Cited by 63

MafB antagonizes phenotypic alteration induced by GM-CSF in microglia

Koshida, R., Oishi, H., Hamada, M., & Takahashi, S. (2015) · Biochemical and Biophysical Research Communications

Neuroinflammation and Neurodegeneration MechanismsImmune cells in cancer

Cited by 20

Ritscher-Schinzel syndrome can be characterized as an endosomal recyclinopathy

Kato, K., Nishio, Y., McMillan, K. J., Al-Maraghi, A., Kroes, H. Y., Abdel-Hamid, M. S., Jones, E., Shaw, S., Yoshida, A., Otsuji, S., et al. (2025) · Science Translational Medicine

Cellular transport and secretionGenomics and Rare Diseases

Cited by 7