Haas, Carola A.

Quiescent and active hippocampal neural stem cells with distinct morphologies respond selectively to physiological and pathological stimuli and aging

Lugert, S., Basak, O., Knuckles, P., Haussler, U., Fabel, K., Götz, M., Haas, C. A., Kempermann, G., Taylor, V., & Giachino, C. (2010) · Cell stem cell

Neurogenesis and neuroplasticity mechanismsDevelopmental Biology and Gene Regulation

Cited by 704

Targeting gene-modified hematopoietic cells to the central nervous system: use of green fluorescent protein uncovers microglial engraftment

Priller, J., Flügel, A., Wehner, T., Boentert, M., Haas, C. A., Prinz, M., Fernández-Klett, F., Prass, K., Bechmann, I., de Boer, B. A., et al. (2001) · Nature Medicine

Virus-based gene therapy researchBarrier Structure and Function Studies

Cited by 637

Astrocyte uncoupling as a cause of human temporal lobe epilepsy

Bedner, P., Dupper, A., Hüttmann, K., Müller, J., Herde, M. K., Dublin, P., Deshpande, T., Schramm, J., Häussler, U., Haas, C. A., et al. (2015) · Brain

Epilepsy research and treatmentConnexins and lens biology

Cited by 328

Induction of Brain-Derived Neurotrophic Factor in Plaque-Associated Glial Cells of Aged APP23 Transgenic Mice

Burbach, G. J., Hellweg, R., Haas, C. A., Del Turco, D., Deicke, U., Abramowski, D., Jucker, M., Staufenbiel, M., & Deller, T. (2004) · The Journal of Neuroscience

Alzheimer's disease research and treatmentsNerve injury and regeneration

Cited by 132

Reelin deficiency causes granule cell dispersion in epilepsy

Haas, C. A., & Frotscher, M. (2010) · Experimental Brain Research

Epilepsy research and treatmentNeurogenesis and neuroplasticity mechanisms

Cited by 96

Functional Inactivation of the Genome-Wide Association Study Obesity Gene Neuronal Growth Regulator 1 in Mice Causes a Body Mass Phenotype

Lee, A. W. S., Hengstler, H., Schwald, K., Berriel-Diaz, M., Loreth, D., Kirsch, M., Kretz, O., Haas, C. A., de Angelis, M. H., Herzig, S., et al. (2012) · PLoS ONE

Regulation of Appetite and ObesityGenetic Associations and Epidemiology

Cited by 84

Epileptiform activity interferes with proteolytic processing of Reelin required for dentate granule cell positioning

Tinnes, S., Schäfer, M. K. E., Flubacher, A., Münzner, G., Frotscher, M., & Haas, C. A. (2011) · The FASEB Journal

Epilepsy research and treatmentNeurogenesis and neuroplasticity mechanisms

Cited by 58

GABA B autoreceptor-mediated cell type-specific reduction of inhibition in epileptic mice

Dugladze, T., Maziashvili, N., Börgers, C., Gurgenidze, S., Häussler, U., Winkelmann, A., Haas, C. A., Meier, J. C., Vida, I., Kopell, N. J., et al. (2013) · Proceedings of the National Academy of Sciences

Epilepsy research and treatmentNeuroscience and Neuropharmacology Research

Cited by 55

TIMP-1 inhibits the proteolytic processing of Reelin in experimental epilepsy

Tinnes, S., Ringwald, J., & Haas, C. A. (2013) · The FASEB Journal

Epilepsy research and treatmentProtease and Inhibitor Mechanisms

Cited by 39

Seizure-induced motility of differentiated dentate granule cells is prevented by the central Reelin fragment

Orcinha, C., Münzner, G., Gerlach, J., Kilias, A., Follo, M., Egert, U., & Haas, C. A. (2016) · Frontiers in Cellular Neuroscience

Epilepsy research and treatmentNeurogenesis and neuroplasticity mechanisms

Cited by 26

Differential vulnerability of neuronal subpopulations of the subiculum in a mouse model for mesial temporal lobe epilepsy

Franz, J., Barheier, N., Wilms, H., Tulke, S., Haas, C. A., & Häussler, U. (2023) · Frontiers in Cellular Neuroscience

Epilepsy research and treatmentNeuroscience and Neuropharmacology Research

Cited by 12