Demontis, Fabio

FOXO/4E-BP Signaling in Drosophila Muscles Regulates Organism-wide Proteostasis during Aging

Demontis, F., & Perrimon, N. (2010) · Cell

Genetics, Aging, and Longevity in Model OrganismsFOXO transcription factor regulation

Cited by 656

The influence of skeletal muscle on systemic aging and lifespan

Demontis, F., Piccirillo, R., Goldberg, A. L., & Perrimon, N. (2013) · Aging Cell

Genetics, Aging, and Longevity in Model OrganismsMuscle Physiology and Disorders

Cited by 238

Mechanisms of skeletal muscle aging: insights from Drosophila and mammalian models

Demontis, F., Piccirillo, R., Goldberg, A. L., & Perrimon, N. (2013) · Disease Models & Mechanisms

Muscle Physiology and DisordersGenetics, Aging, and Longevity in Model Organisms

Cited by 238

Skeletal muscle autophagy and its role in sarcopenia and organismal aging

Jiao, J., & Demontis, F. (2017) · Current Opinion in Pharmacology

Muscle Physiology and DisordersAutophagy in Disease and Therapy

Cited by 184

Systemic Nutrient and Stress Signaling via Myokines and Myometabolites

Rai, M., & Demontis, F. (2016) · Annual Review of Physiology

Muscle Physiology and DisordersExercise and Physiological Responses

Cited by 123

A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice

Hunt, L. C., Stover, J., Haugen, B., Shaw, T. I., Li, Y., Pagala, V. R., Finkelstein, D., Barton, E. R., Fan, Y., Labelle, M., et al. (2019) · Cell Reports

Muscle Physiology and DisordersUbiquitin and proteasome pathways

Cited by 90

Contribution of proteases to the hallmarks of aging and to age-related neurodegeneration

Rai, M., Curley, M., Coleman, Z., & Demontis, F. (2022) · Aging Cell

Genetics, Aging, and Longevity in Model OrganismsProtease and Inhibitor Mechanisms

Cited by 72

The glucose-sensing transcription factor MLX promotes myogenesis via myokine signaling

Hunt, L. C., Xu, B., Finkelstein, D., Fan, Y., Carroll, P. A., Cheng, P.-F., Eisenman, R. N., & Demontis, F. (2015) · Genes & Development

Muscle Physiology and DisordersMetabolism, Diabetes, and Cancer

Cited by 64