Marti, Thomas M.
University of Bern · University Hospital of Bern
5 papers on CoChatORCID 0000-0003-3005-220X
Most cited papers
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H2AX phosphorylation within the G 1 phase after UV irradiation depends on nucleotide excision repair and not DNA double-strand breaks
Marti, T. M., Hefner, E., Feeney, L., Natale, V., & Cleaver, J. E. (2006) · Proceedings of the National Academy of Sciences
DNA Repair MechanismsEffects of Radiation Exposure
Cited by 359
- Verified
A microfluidic platform for chemoresistive testing of multicellular pleural cancer spheroids
Ruppen, J., Cortes-Dericks, L., Marconi, E., Karoubi, G., Schmid, R. A., Peng, R., Marti, T. M., & Guenat, O. T. (2014) · Lab Chip
3D Printing in Biomedical ResearchMicrofluidic and Capillary Electrophoresis Applications
Cited by 105
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HSP90/AXL/eIF4E-regulated unfolded protein response as an acquired vulnerability in drug-resistant KRAS-mutant lung cancer
Yang, H., Liang, S.-Q., Xu, D., Yang, Z., Marti, T. M., Gao, Y., Kocher, G. J., Zhao, H., Schmid, R. A., & Peng, R.-W. (2019) · Oncogenesis
Lung Cancer Treatments and MutationsEndoplasmic Reticulum Stress and Disease
Cited by 58
- Verified
mTOR mediates a mechanism of resistance to chemotherapy and defines a rational combination strategy to treat KRAS-mutant lung cancer
Liang, S.-Q., Bührer, E. D., Berezowska, S., Marti, T. M., Xu, D., Froment, L., Yang, H., Hall, S. R. R., Vassella, E., Yang, Z., et al. (2019) · Oncogene
PI3K/AKT/mTOR signaling in cancerLung Cancer Treatments and Mutations
Cited by 55
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Non-genetic adaptive resistance to KRASG12C inhibition: EMT is not the only culprit
Ning, W., Marti, T. M., Dorn, P., & Peng, R.-W. (2022) · Frontiers in Oncology
Cancer Cells and MetastasisLung Cancer Treatments and Mutations
Cited by 29
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