Abraham, Clara

Host–microbe interactions have shaped the genetic architecture of inflammatory bowel disease

The International IBD Genetics Consortium (IIBDGC), Jostins, L., Ripke, S., Weersma, R. K., Duerr, R. H., McGovern, D. P., Hui, K. Y., Lee, J. C., Philip Schumm, L., Sharma, Y., et al. (2012) · Nature

Inflammatory Bowel DiseaseGenetic Associations and Epidemiology

Cited by 4,722

A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene

Duerr, R. H., Taylor, K. D., Brant, S. R., Rioux, J. D., Silverberg, M. S., Daly, M. J., Steinhart, A. H., Abraham, C., Regueiro, M., Griffiths, A., et al. (2006) · Science

Inflammatory Bowel DiseaseGenetic Associations and Epidemiology

Cited by 3,039

Inflammatory bowel disease

Abraham, C., & Cho, J. H. (2009) · New England Journal of Medicine

Inflammatory Bowel DiseaseOcular Diseases and Behçet’s Syndrome

Cited by 2,712

Targeted epithelial tight junction dysfunction causes immune activation and contributes to development of experimental colitis

Su, L., Shen, L., Clayburgh, D. R., Nalle, S. C., Sullivan, E. A., Meddings, J. B., Abraham, C., & Turner, J. R. (2009) · Gastroenterology

Inflammatory Bowel DiseaseGut microbiota and health

Cited by 455

LACC1 Required for NOD2-Induced, ER Stress-Mediated Innate Immune Outcomes in Human Macrophages and LACC1 Risk Variants Modulate These Outcomes

Huang, C., Hedl, M., Ranjan, K., & Abraham, C. (2019) · Cell Reports

Endoplasmic Reticulum Stress and DiseaseImmune Response and Inflammation

Cited by 30