Wooten, Marie W.

Sequestosome 1/p62 shuttles polyubiquitinated tau for proteasomal degradation

Babu, J. R., Geetha, T., & Wooten, M. W. (2005) · Journal of Neurochemistry

Alzheimer's disease research and treatmentsUbiquitin and proteasome pathways

Cited by 315

Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration

Ramesh Babu, J., Lamar Seibenhener, M., Peng, J., Strom, A., Kemppainen, R., Cox, N., Zhu, H., Wooten, M. C., Diaz‐Meco, M. T., Moscat, J., et al. (2008) · Journal of Neurochemistry

Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research

Cited by 258

Sequestosome 1/p62 links familial ALS mutant SOD1 to LC3 via an ubiquitin‐independent mechanism

Gal, J., Ström, A., Kwinter, D. M., Kilty, R., Zhang, J., Shi, P., Fu, W., Wooten, M. W., & Zhu, H. (2009) · Journal of Neurochemistry

Amyotrophic Lateral Sclerosis ResearchAutophagy in Disease and Therapy

Cited by 187

Oxidative damage to the promoter region of SQSTM1/p62 is common to neurodegenerative disease

Du, Y., Wooten, M. C., & Wooten, M. W. (2009) · Neurobiology of Disease

Alzheimer's disease research and treatmentsRedox biology and oxidative stress

Cited by 96

Behavioral effects of SQSTM1/p62 overexpression in mice: Support for a mitochondrial role in depression and anxiety

Seibenhener, M. L., Zhao, T., Du, Y., Calderilla-Barbosa, L., Yan, J., Jiang, J., Wooten, M. W., & Wooten, M. C. (2013) · Behavioural Brain Research

Mitochondrial Function and PathologyTreatment of Major Depression

Cited by 22