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Cited by 172

The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.

THE LIVER IN OBESITY

ZELMAN, SAMUEL·1952·Archives of Internal Medicine

Abstract

EXPERIMENTAL obesity produced by hypothalamic injury in various species of animals is uniformly accompanied with liver damage, progressing occasionally to cirrhosis.1In hereditarily obese strains of mice, liver damage is also seen uniformly, and even tumors of the liver are observed.2Although the increased incidence in human obesity of gall-bladder disease3and diabetes mellitus4has long been known, and although these conditions may lead independently to liver disease, there does not appear to be in the literature a consideration of the existence of liver damage in obesity per se. The majority of obese persons show a significant decrease in carbohydrate tolerance,5and this impairment has been related to the duration rather than to the degree of obesity.6Similar decreases in carbohydrate tolerance are observed in the experimental obesity of hypothalamic injury1and in hereditarily obese mice.7Newburgh and Conn8suggested that the disturbed carbohydrate metabolism occurring with obesity is related

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Metadata

Authors
ZELMAN, S.
Venue
Archives of Internal Medicine
Published
1952-08-01
Volume
90
Issue
2
Pages
141
ISSN
0003-9926 (ISSN-L)
OpenAlex
W2121332447
Cited by
172
References
1
FWCI
12.54
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: THE LIVER IN OBESITY. https://app.cochat.ai/papers/10.1001/archinte.1952.00240080007002
The paper
ZELMAN, S. (1952). THE LIVER IN OBESITY. Archives of Internal Medicine. https://doi.org/10.1001/archinte.1952.00240080007002

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.