Using PU.1 and Jun dimerization protein 2 transcription factor expression in myelodysplastic syndromes to predict treatment response and leukaemia transformation
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Several other transcriptional regulators interact with PU.1, including AP-1 family transcription factors.
“Moreover, several other transcriptional regulators interact with PU.1, including AP-1 family transcription factors (Steidl et al., 2006; Boasman et al., 2019; Zhao et al., 2022).”
From PU.1 is required to restrain myelopoiesis during chronic inflammatory stress (2023) · cites this paper as Boasman et al., 2019
Cites
Bagger, F. O., Sasivarevic, D., Sohi, S. H., Laursen, L. G., Pundhir, S., Sønderby, C. K., Winther, O., Rapin, N., & Porse, B. T. (2015) · Nucleic Acids Research · doi:10.1093/nar/gkv1101 · Cited by 330 · see citing works
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Self-citationEVI1 Impairs Myelopoiesis by Deregulation of PU.1 Function
Laricchia‐Robbio, L., Premanand, K., Rinaldi, C. R., & Nucifora, G. (2009) · Cancer Research · doi:10.1158/0008-5472.can-08-2562 · Cited by 74 · see citing works
Self-citationCurik, N., Burda, P., Vargová, K., Pospíšil, V., Beličková, M., Vlckova, P., Savvulidi, F. G., Nečas, E., Hajkova, H., Haškovec, C., et al. (2012) · Leukemia · doi:10.1038/leu.2012.47 · Cited by 45 · see citing works
Metallothionein-1 Isoforms and Vimentin Are Direct PU.1 Downstream Target Genes in Leukemia Cells
Imoto, A., Okada, M., Okazaki, T., Kitasato, H., Harigae, H., & Takahashi, S. (2010) · Journal of Biological Chemistry · doi:10.1074/jbc.m109.095810 · Cited by 28 · see citing works
van der Weyden, L., Rust, A. G., McIntyre, R. E., Robles‐Espinoza, C. D., Velasco‐Herrera, M. D. C., Strogantsev, R., Ferguson‐Smith, A. C., McCarthy, S., Keane, T., Arends, M. J., et al. (2012) · Oncogene · doi:10.1038/onc.2012.56 · Cited by 24 · see citing works
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Haider, M., Duncavage, E. J., Afaneh, K. F., Bejar, R. L., & List, A. F. (2017) · American Society of Clinical Oncology Educational Book · doi:10.1200/edbk_175397 · Cited by 16 · see citing works
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Unravelling the Epigenome of Myelodysplastic Syndrome: Diagnosis, Prognosis, and Response to Therapy
Bond, D. R., Lee, H. J., & Enjeti, A. K. (2020) · Cancers · doi:10.3390/cancers12113128 · Cited by 25 · see citing works
PU.1 is required to restrain myelopoiesis during chronic inflammatory stress
Chavez, J. S., Rabe, J. L., Niño, K. E., Wells, H. H., Gessner, R. L., Mills, T. S., Hernandez, G., & Pietras, E. M. (2023) · Frontiers in Cell and Developmental Biology · doi:10.3389/fcell.2023.1204160 · Cited by 14 · see citing works
“1, including AP-1 family transcription factors (Steidl et al., 2006; Boasman et al., 2019; Zhao et al., 2022).”
ANP32B suppresses B‐cell acute lymphoblastic leukemia through activation of PU .1 in mice
Yang, Q., Liu, H., Yang, S., Wei, Y., Zhu, X., Zhi, Z., Zhu, D., Chen, G., & Yu, Y. (2023) · Cancer Science · doi:10.1111/cas.15822 · Cited by 11 · see citing works
Predicting outcome in higher-risk myelodysplastic syndrome patients treated with azacitidine
Bouchla, A., Thomopoulos, T. P., Papageorgiou, S. G., Apostolopoulou, C. A., Loucari, C. C., Mpazani, E., & Pappa, V. (2021) · Epigenomics · doi:10.2217/epi-2021-0124 · Cited by 4 · see citing works
Jabbar, S., & Ismael, A. (2023) · Iraqi Journal of Cancer and Medical Genetics · doi:10.29409/ijcmg.v16i2.338 · Cited by 2 · see citing works
- Authors
- Boasman, K., Simmonds, M. J., Graham, C., Saunthararajah, Y., & Rinaldi, C. R.
- Affiliations
- University of Lincoln (GB)
- Queen's University Belfast (GB)
- Cleveland Clinic (US)
- Corresponding author
- Rinaldi, Ciro Roberto
- Venue
- Annals of Hematology
- Published
- 2019-02-05
- Volume
- 98
- Issue
- 6
- Pages
- 1529-1531
- ISSN
- 0939-5555 (ISSN-L)
- 1432-0584
- OpenAlex
- W2914481845
- Cited by
- 5
- References
- 8
- Citations by year
- 2023: 3 · 2021: 1 · 2020: 1
- FWCI
- 0.36
- Type
- journal article
- Language
- en
- Open access
- closed
- Licence
- www.springer.com/tdm (tdm)
- Locations
- Annals of Hematology (publishedVersion)
- PubMed (publishedVersion)
- Lincoln Repository (University of Lincoln) (acceptedVersion)
- Funders
- Celgene (NA)
- Topics
- Health Sciences › Medicine › Hematology › Acute Myeloid Leukemia Research
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Retinoids in leukemia and cellular processes
- Health Sciences › Medicine › Hematology › Hematopoietic Stem Cell Transplantation
- Keywords
- PU.1, MyD88, transcription factor expression, myelodysplastic syndrome, treatment response prediction, prognosis
- MeSH terms
- Proto-Oncogene Protein Spi-1, Antimetabolites, Antineoplastic, Azacitidine, Bone Marrow, Cell Transformation, Neoplastic, Humans, Myelodysplastic Syndromes, Prognosis, Proto-Oncogene Proteins, Repressor Proteins, Biomarkers, Tumor, Biomarkers, Leukemia, Myeloid, Acute, Trans-Activators, Down-Regulation, Up-Regulation, Gene Expression Regulation, Neoplastic, Disease Progression, DNA Methylation
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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