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Cited by 134

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Curcumin attenuates inflammation through inhibition of TLR-4 receptor in experimental colitis

Lubbad, A., Oriowo, M. A., Khan, I.·2009·Molecular and Cellular Biochemistry
DOI 10.1007/s11010-008-9949-4
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Show all 2 references

Research works printed but not linked: 21

  1. Schmidt C, Stallmach A (2005) Etiology and pathogenesis of inflammatory bowel disease. Minerva Gastroenterol Dietol 51(2):127–145
  2. Atreya I, Atreya R, Neurath MF (2008) NF-kappaB in inflammatory bowel disease. J Intern Med 263(6):591–596. doi: 10.1111/j.1365-2796.2008.01953.x (Review) · DOI · Paper page
  3. Kaser A, Tilg H (2008) Novel therapeutic targets in the treatment of IBD. Expert Opin Ther Targets 12(5):553–563. doi: 10.1517/14728222.12.5.553 · DOI
  4. Hanauer SB (1996) Inflammatory bowel disease. N Engl J Med 334(13):841–848. doi: 10.1056/NEJM199603283341307 (Review) · DOI
  5. Pari L, Tewas D, Eckel J (2008) Role of curcumin in health and disease. Arch Physiol Biochem 114(2):127–149. doi: 10.1080/13813450802033958 (Review) · DOI
  6. Kozuch PL, Hanauer SB (2008) Treatment of inflammatory bowel disease: a review of medical therapy. World J Gastroenterol 14(3):354–377. doi: 10.3748/wjg.14.354 (Review) · DOI · Paper page
  7. Billerey-Larmonier C, Uno JK, Larmonier N, Midura AJ, Timmermann B, Ghishan FK, Kiela PR (2008) Protective effects of dietary curcumin in mouse model of chemically induced colitis are strain dependent. Inflamm Bowel Dis 14(6):780–793. doi: 10.1002/ibd.20348 · DOI
  8. Holt PR, Katz S, Kirshoff R (2005) Curcumin therapy in inflammatory bowel disease: a pilot study. Dig Dis Sci 50(11):2191–2193. doi: 10.1007/s10620-005-3032-8 · DOI
  9. Boone DL, Ma A (2003) Connecting the dots from toll-like receptors to innate immune cells and inflammatory bowel disease. J Clin Invest 111(9):1284–1286 · DOI
  10. Bou-Fersen AM, Anim JT, Khan I (2008) Experimental colitis is associated with ultrastructural changes in inflamed and uninflamed regions of the gastrointestinal tract. Med Princ Pract 17(3):190–196. doi: 10.1159/000117791 · DOI
  11. Al-Jarallah A, Khan I, Oriowo MA (2008) Role of Ca2+-sensitization in attenuated carbachol-induced contraction of the colon in a rat model of colitis. Eur J Pharmacol 579(1–3):365–373. doi: 10.1016/j.ejphar.2007.10.069 · DOI
  12. Morris GP, Beck PL, Herridge MS, Depew WT, Szewczuk MR, Wallace JL (1989) Hapten-induced model of chronic inflammation and ulceration in the rat colon. Gastroenterology 96(3):795–803 · DOI
  13. Khan I, Oriowo MA, Anim JT (2005) Amelioration of experimental colitis by Na–H exchanger-1 inhibitor amiloride is associated with reversal of IL-1ss and ERK mitogen-activated protein kinase. Scand J Gastroenterol 40(5):578–585. doi: 10.1080/00365520510012352 · DOI
  14. Khan I (2002) Antisense inhibition of cyclooxygenase-2 causes a selective suppression of the Na+–H+ exchanger isoform 3 in rat kidney in experimental colitis. Nephron 91(1):120–128. doi: 10.1159/000057613 · DOI
  15. Bradley PP, Priebat DA, Christensen RD, Rothstein G (1982) Measurement of cutaneous inflammation: estimation of neutrophil content with an enzyme marker. J Invest Dermatol 78(3):206–209. doi: 10.1111/1523-1747.ep12506462 · DOI
  16. Khan I, Al-Awadi FM, Thomas N (2001) In vivo inhibition of cyclooxygenase-2 by a selective phosphorothioated oligonucleotide. Antisense Nucleic Acid Drug Dev 11(4):199–207. doi: 10.1089/108729001317022205 · DOI
  17. Wang K, Wan YJ (2008) Nuclear receptors and inflammatory diseases. Exp Biol Med (Maywood) 233(5):496–506. doi: 10.3181/0708-MR-231 (Review) · DOI
  18. Hogaboam CM, Vallance BA, Kumar A, Addison CL, Graham FL, Gauldie J, Collins SM (1997) Therapeutic effects of interleukin-4 gene transfer in experimental inflammatory bowel disease. J Clin Invest 100(11):2766–2776. doi: 10.1172/JCI119823 · DOI
  19. Arranz A, Juarranz Y, Leceta J, Gomariz RP, Martínez C (2008) VIP balances innate and adaptive immune responses induced by specific stimulation of TLR2 and TLR4. Peptides 29(6):948–956. doi: 10.1016/j.peptides.2008.01.019 · DOI
  20. Arranz A, Abad C, Juarranz Y, Torroba M, Rosignoli F, Leceta J, Gomariz RP, Martínez C (2006) Effect of VIP on TLR2 and TLR4 expression in lymph node immune cells during TNBS-induced colitis. Ann N Y Acad Sci 1070:129–134. doi: 10.1196/annals.1317.001 · DOI
  21. Araki A, Kanai T, Ishikura T, Makita S, Uraushihara K, Iiyama R, Totsuka T, Takeda K, Akira S, Watanabe M (2005) MyD88-deficient mice develop severe intestinal inflammation in dextran sodium sulfate colitis. J Gastroenterol 40:16–23. doi: 10.1007/s00535-004-1492-9 · DOI

Cited by

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Metadata

Authors
Lubbad, A., Oriowo, M. A., & Khan, I.
Venue
Molecular and Cellular Biochemistry
Publisher
Springer Science and Business Media LLC
Published
2008-11-11
Volume
322
Issue
1-2
Pages
127-135
ISSN
  • 0300-8177
  • 1573-4919
DOI
10.1007/s11010-008-9949-4
Cited by
134
References
2
Language
en
Licence
www.springer.com/tdm (tdm)

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CoChat. (2026). Citation check: Curcumin attenuates inflammation through inhibition of TLR-4 receptor in experimental colitis. https://app.cochat.ai/papers/10.1007/s11010-008-9949-4
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Lubbad, A., Oriowo, M. A., & Khan, I. (2009). Curcumin attenuates inflammation through inhibition of TLR-4 receptor in experimental colitis. Molecular and Cellular Biochemistry. https://doi.org/10.1007/s11010-008-9949-4

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.