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Cited by 371

The DOI matches its Crossref record; 263 of the 263 references it cites were found in a registry or an index.

Pathogenesis, diagnostics, and therapeutics for Alzheimer's disease: Breaking the memory barrier

Open access
Kamatham, Pushpa Tryphena, Shukla, Rashi, Khatri, Dharmendra Kumar, Vora, Lalitkumar K.·2024·Ageing Research Reviews

Abstract

Alzheimer's disease (AD) is the most common cause of dementia and accounts for 60-70 % of all cases. It affects millions of people worldwide. AD poses a substantial economic burden on societies and healthcare systems. AD is a progressive neurodegenerative disorder characterized by cognitive decline, memory loss, and impaired daily functioning. As the prevalence of AD continues to increase, understanding its pathogenesis, improving diagnostic methods, and developing effective therapeutics have become paramount. This comprehensive review delves into the intricate mechanisms underlying AD, explores the current state of diagnostic techniques, and examines emerging therapeutic strategies. By revealing the complexities of AD, this review aims to contribute to the growing body of knowledge surrounding this devastating disease.

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Research works printed but not linked: 21

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Metadata

Authors
Kamatham, P. T., Shukla, R., Khatri, D. K., & Vora, L. K.
Venue
Ageing Research Reviews
Publisher
Elsevier BV
Published
2024-11
Volume
101
Pages
102481
ISSN
1568-1637 (ISSN-L)
OpenAlex
W4402169022
Cited by
371
References
263
FWCI
28.43
Type
journal article
Language
en
Open access
hybrid
Licence
Funders
National Institute of Pharmaceutical Education and Research, Raebareli

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CoChat. (2026). Citation check: Pathogenesis, diagnostics, and therapeutics for Alzheimer's disease: Breaking the memory barrier. https://app.cochat.ai/papers/10.1016/j.arr.2024.102481
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Kamatham, P. T., Shukla, R., Khatri, D. K., & Vora, L. K. (2024). Pathogenesis, diagnostics, and therapeutics for Alzheimer's disease: Breaking the memory barrier. Ageing Research Reviews. https://doi.org/10.1016/j.arr.2024.102481

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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