The DOI matches its Crossref record; 17 of the 17 references it cites were found in a registry or an index.
Anti-apoptosis and cell survival: A review
Open accessAbstract
Type I programmed cell death (PCD) or apoptosis is critical for cellular self-destruction for a variety of processes such as development or the prevention of oncogenic transformation. Alternative forms, including type II (autophagy) and type III (necrotic) represent the other major types of PCD that also serve to trigger cell death. PCD must be tightly controlled since disregulated cell death is involved in the development of a large number of different pathologies. To counter the multitude of processes that are capable of triggering death, cells have devised a large number of cellular processes that serve to prevent inappropriate or premature PCD. These cell survival strategies involve a myriad of coordinated and systematic physiological and genetic changes that serve to ward off death. Here we will discuss the different strategies that are used to prevent cell death and focus on illustrating that although anti-apoptosis and cellular survival serve to counteract PCD, they are nevertheless mechanistically distinct from the processes that regulate cell death.
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Cites
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Discovery of immune molecules and their crucial functions in shrimp immunity
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Albendazole as a promising molecule for tumor control
Castro, L. S. E. P. W., Kviecinski, M. R., Ourique, F., Parisotto, E. B., Grinevicius, V. M. A. S., Correia, J. F. G., Wilhelm Filho, D., & Pedrosa, R. C. (2016) · Redox Biology · doi:10.1016/j.redox.2016.09.013 · Cited by 81
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Metadata
- Authors
- Portt, L., Norman, G., Clapp, C., Greenwood, M., & Greenwood, M. T.
- Venue
- Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Publisher
- Elsevier BV
- Published
- 2011-01
- Volume
- 1813
- Issue
- 1
- Pages
- 238-259
- ISSN
- 0167-4889 (ISSN-L)
- OpenAlex
- W2099818615
- Cited by
- 636
- References
- 17
- FWCI
- 7.33
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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