Bipolar Disorder as a Mitochondrial Disease
Integrity signals
Registry
Cites
Statistics
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
Bipolar disorder is characterized by mutations to mtDNA and mitochondrial dysfunction caused predominantly by excess ROS.
“Bipolar disorder is also characterized by mutations to mtDNA and mitochondrial dysfunction caused predominantly by excess ROS (Andreazza et al. 2018; Yager et al. 2010).”
From Reactive Oxygen Species, Toxicity, Oxidative Stress, and Antioxidants: Chronic Diseases and Aging (2023) · cites this paper as Andreazza et al. 2018
Cites
Stork, C., & Renshaw, P. F. (2005) · Molecular Psychiatry · doi:10.1038/sj.mp.4001711 · Cited by 486 · see citing works
Andreazza, A. C., Shao, L., Wang, J.-F., & Young, L. T. (2010) · Archives of General Psychiatry · doi:10.1001/archgenpsychiatry.2010.22 · Cited by 448 · see citing works
Self-citationMutant NDUFV1 subunit of mitochondrial complex I causes leukodystrophy and myoclonic epilepsy
Schuelke, M., Smeitink, J. A. M., Mariman, E. C. M., Loeffen, J. L. C., Plecko, B., Trijbels, F. J. M., Stöckler‐Ipsiroglu, S., & van den Heuvel, L. (1999) · Nature Genetics · doi:10.1038/6772 · Cited by 270 · see citing works
The Psychiatric Manifestations of Mitochondrial Disorders: A Case and Review of the Literature
Anglin, R. E., Garside, S. L., Tarnopolsky, M. A., Mazurek, M. F., & Rosebush, P. I. (2012) · The Journal of Clinical Psychiatry · doi:10.4088/jcp.11r07237 · Cited by 150 · see citing works
Mitochondrial DNA 3644T→C mutation associated with bipolar disorder
Munakata, K., Tanaka, M., Mori, K., Washizuka, S., Yoneda, M., Tajima, O., Akiyama, T., Nanko, S., Kunugi, H., Tadokoro, K., et al. (2004) · Genomics · doi:10.1016/j.ygeno.2004.08.015 · Cited by 113 · see citing works
Yoshimi, N., Futamura, T., Bergen, S. E., Iwayama, Y., Ishima, T., Sellgren, C. M., Ekman, C. J., Jakobsson, J., Pålsson, E., Kakumoto, K., et al. (2016) · Molecular Psychiatry · doi:10.1038/mp.2015.217 · Cited by 110 · see citing works
Scola, G., Kim, H. K., Young, L. T., & Andreazza, A. C. (2012) · Biological Psychiatry · doi:10.1016/j.biopsych.2012.06.028 · Cited by 66 · see citing works
Self-citationLi, C., Bai, Y., Hsieh, J., Lee, H., Yang, B., Chen, M., Lin, W.-C., Tsai, C.-F., Tu, P., Wang, S.-J., et al. (2015) · Psychoneuroendocrinology · doi:10.1016/j.psyneuen.2015.02.003 · Cited by 21 · see citing works
Frye, M. A., Ryu, E., Nassan, M. M., Jenkins, G. D., Andreazza, A. C., Evans, J. M., McElroy, S. L., Oglesbee, D., Highsmith, W. E., & Biernacka, J. M. (2016) · Journal of Psychiatric Research · doi:10.1016/j.jpsychires.2016.09.027 · Cited by 19 · see citing works
Self-citationDoyle, G. A. R., Dahl, J. P., Bloch, P. J., et al. (2010) · Psychiatric Genetics · doi:10.1097/ypg.0b013e328341333f · Cited by 11 · see citing works
Flagged first, then by citations
Cited by
Sorted by citations
Reactive Oxygen Species, Toxicity, Oxidative Stress, and Antioxidants: Chronic Diseases and Aging
Jomova, K., Raptova, R., Alomar, S. Y., Alwasel, S. H., Nepovimova, E., Kuca, K., & Valko, M. (2023) · Archives of Toxicology · doi:10.1007/s00204-023-03562-9 · Cited by 2,638 · see citing works
McIntyre, R. S., Berk, M., Brietzke, E., Goldstein, B. I., López-Jaramillo, C., Kessing, L. V., Malhi, G. S., Nierenberg, A. A., Rosenblat, J. D., Majeed, A., et al. (2020) · The Lancet · doi:10.1016/s0140-6736(20)31544-0 · Cited by 1,047 · see citing works
- Authors
- Andreazza, A. C., Duong, A., & Young, L. T.
- Affiliations
- Centre for Addiction and Mental Health (CA)
- University of Toronto (CA)
- Corresponding author
- Andreazza, Ana C.
- Venue
- Biological Psychiatry
- Publisher
- Elsevier BV
- Published
- 2018-05
- Volume
- 83
- Issue
- 9
- Pages
- 720-721
- ISSN
- 0006-3223 (ISSN-L)
- OpenAlex
- W2757494765
- Cited by
- 49
- References
- 10
- Citations by year
- 2026: 4 · 2025: 9 · 2024: 4 · 2023: 7 · 2022: 7 · 2021: 9 · 2020: 2 · 2019: 2 · 2018: 4 · 2017: 1
- FWCI
- 2.03
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Biological Psychiatry (publishedVersion)
- PubMed (publishedVersion)
- Funders
- Canadian Institutes of Health Research (MOP133439, MOP-484414)
- Topics
- Health Sciences › Medicine › Psychiatry and Mental health › Bipolar Disorder and Treatment
- Health Sciences › Medicine › Pharmacology › Treatment of Major Depression
- Health Sciences › Medicine › Psychiatry and Mental health › Schizophrenia research and treatment
- Keywords
- bipolar disorder, mitochondrial disease, mitochondrial dysfunction, relapse, psychosis, biomarkers, diagnosis, treatment, health care costs
- MeSH terms
- Bipolar Disorder, DNA, Mitochondrial, Humans, Mitochondrial Diseases
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review