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Cited by 417

The DOI matches its Crossref record; 90 of the 90 references it cites were found in a registry or an index.

Zika Virus Pathogenesis and Tissue Tropism

Open access
Miner, Jonathan J., Diamond, Michael S.·2017·Cell Host & Microbe

Abstract

Although Zika virus (ZIKV) was isolated approximately 70 years ago, few experimental studies had been published prior to 2016. The recent spread of ZIKV to countries in the Western Hemisphere is associated with reports of microcephaly, congenital malformations and Guillain-Barré syndrome. This has resulted in ZIKV being declared a public health emergency and has greatly accelerated the pace of ZIKV research and discovery. Within a short time period, useful mouse and non-human primate disease models have been established, and pre-clinical evaluation of therapeutics and vaccines has begun. Unexpectedly, ZIKV exhibits a broad tropism and persistence in body tissues and fluids, which contributes to the clinical manifestations and epidemiology that have been observed during the current epidemic. In this review, we highlight recent advances in our understanding of ZIKV pathogenesis, tissue tropism and the resulting pathology, and discuss areas for future investigation.

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Metadata

Authors
Miner, J. J., & Diamond, M. S.
Venue
Cell Host & Microbe
Publisher
Elsevier BV
Published
2017-02
Volume
21
Issue
2
Pages
134-142
ISSN
1931-3128 (ISSN-L)
OpenAlex
W2586860795
Cited by
417
References
90
FWCI
48.78
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Zika Virus Pathogenesis and Tissue Tropism. https://app.cochat.ai/papers/10.1016/j.chom.2017.01.004
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Miner, J. J., & Diamond, M. S. (2017). Zika Virus Pathogenesis and Tissue Tropism. Cell Host & Microbe. https://doi.org/10.1016/j.chom.2017.01.004

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.