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Cited by 332

The DOI matches its Crossref record; 161 of the 161 references it cites were found in a registry or an index.

Manganese neurotoxicity and the role of reactive oxygen species

Open access
Martinez-Finley, Ebany J., Gavin, Claire E., Aschner, Michael, Gunter, Thomas E.·2013·Free Radical Biology and Medicine

Abstract

Manganese (Mn) is an essential dietary nutrient but excess or accumulations can be toxic. Disease states, like manganism, are associated with overexposure or accumulation of Mn and are due to the production of reactive oxygen species, free radicals and toxic metabolites, alteration of mitochondrial function and ATP production and depletion of cellular antioxidant defense mechanisms. This review focuses on all of the preceding mechanisms and the scientific studies that support them as well as provides an overview of the absorption, distribution, and excretion of Mn and the stability and transport of Mn compounds in the body.

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Crossref · 10.1016/j.freeradbiomed.2013.01.032

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Metadata

Authors
Martinez-Finley, E. J., Gavin, C. E., Aschner, M., & Gunter, T. E.
Venue
Free Radical Biology and Medicine
Publisher
Elsevier BV
Published
2013-09
Volume
62
Pages
65-75
ISSN
0891-5849 (ISSN-L)
OpenAlex
W2009132341
Cited by
332
References
161
FWCI
16.11
Type
journal article
Language
en
Open access
green

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CoChat. (2026). Citation check: Manganese neurotoxicity and the role of reactive oxygen species. https://app.cochat.ai/papers/10.1016/j.freeradbiomed.2013.01.032
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Martinez-Finley, E. J., Gavin, C. E., Aschner, M., & Gunter, T. E. (2013). Manganese neurotoxicity and the role of reactive oxygen species. Free Radical Biology and Medicine. https://doi.org/10.1016/j.freeradbiomed.2013.01.032

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.