The DOI matches its Crossref record; 15 of the 15 references it cites were found in a registry or an index.
Innate Lymphoid Cell Interactions with Microbiota: Implications for Intestinal Health and Disease
Open accessAbstract
The mammalian intestine harbors trillions of beneficial commensal bacteria that are essential for the development of the immune system and for maintenance of physiologic processes in multiple organs. However, numerous chronic infectious, inflammatory and metabolic diseases in humans have been associated with alterations in the composition or localization of commensal bacteria that results in dysregulated host-commensal bacteria relationships. The mammalian immune system plays an essential role in regulating the acquisition, composition and localization of commensal bacterial communities in the intestine. Emerging research has implicated innate lymphoid cells (ILCs) as a critical immune cell population that orchestrates some of these host-commensal relationships that can impact immunity, inflammation and tissue homeostasis in the intestine. This review will discuss reciprocal interactions between intestinal commensal bacteria and ILCs in the context of health and disease.
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Cites
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Cited by
Role of the normal gut microbiota
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Self-citationRole of the gut microbiota in immunity and inflammatory disease
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The microbiome and innate immunity
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Dysbiosis and the immune system
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Metadata
- Authors
- Sonnenberg, G. F., & Artis, D.
- Venue
- Immunity
- Publisher
- Elsevier BV
- Published
- 2012-10
- Volume
- 37
- Issue
- 4
- Pages
- 601-610
- ISSN
- 1074-7613 (ISSN-L)
- OpenAlex
- W2023731313
- Cited by
- 277
- References
- 15
- FWCI
- 12.08
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Sonnenberg, G. F., & Artis, D. (2012). Innate Lymphoid Cell Interactions with Microbiota: Implications for Intestinal Health and Disease. Immunity. https://doi.org/10.1016/j.immuni.2012.10.003
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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