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Identification of a Novel Dioxin-Inducible Cytochrome P450
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“This receptor was also more highly expressed in Hepa1–6, providing a ready explanation for the up-regulation.”
From Comparative proteomic phenotyping of cell lines and primary cells to assess preservation of cell type-specific functions (2009) · cites this paper as [26]
In the citing paper
“Two of them (CYP1A1 and CYP2S1) are known to be regulated by the aryl hydrogen receptor (25, 26).”
From Comparative proteomic phenotyping of cell lines and primary cells to assess preservation of cell type-specific functions (2009) · cites this paper as [26]
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“Dioxin is the most carcinogenic substance evaluated (Hankinson, 1995).”
Self-citationINDUCTION OF CYTOCHROME P4501A1
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Characterization and expression of extrahepatic CYP2S1
Deb, S., & Bandiera, S. M. (2009) · Expert Opinion on Drug Metabolism & Toxicology · doi:10.1517/17425250902865586 · Cited by 15
Metadata
- Authors
- Rivera, S. P., Saarikoski, S. T., & Hankinson, O.
- Affiliations
- University of California, Los Angeles (US)
- Venue
- Molecular Pharmacology
- Publisher
- Elsevier BV
- Published
- 2002-02
- Volume
- 61
- Issue
- 2
- Pages
- 255-259
- ISSN
- 0026-895X (ISSN-L)
- OpenAlex
- W2166454597
- Cited by
- 149
- References
- 12
- Citations by year
- 2025: 3 · 2024: 3 · 2022: 4 · 2021: 4 · 2020: 3 · 2019: 4 · 2017: 5 · 2015: 5 · 2014: 3 · 2013: 5 · 2012: 6
- FWCI
- 12.50
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Molecular Pharmacology (publishedVersion)
- PubMed (publishedVersion)
- Funders
- National Cancer Institute (2 R01 CA28868-20, R01CA028868, R01 CA 28868)
- Topics
- Life Sciences › Pharmacology, Toxicology and Pharmaceutics › Pharmacology › Pharmacogenetics and Drug Metabolism
- Physical Sciences › Environmental Science › Health, Toxicology and Mutagenesis › Toxic Organic Pollutants Impact
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Peroxisome Proliferator-Activated Receptors
- Keywords
- CYP2S1, cytochrome P450, 2,3,7,8-tetrachlorodibenzo-p-dioxin, aryl hydrocarbon receptor, gene induction, lung epithelial cells, liver, representational difference analysis, cDNA cloning
- MeSH terms
- Amino Acid Sequence, Animals, Carcinogens, Cell Transformation, Neoplastic, Cytochrome P-450 Enzyme System, Dioxins, Enzyme Induction, Humans, Molecular Sequence Data, Oxygenases, RNA, Messenger, Transfection, Tumor Cells, Cultured, Gene Expression, Sequence Homology, Amino Acid, Mice
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