Cited by 27

Chapter 13 Neurotrauma/neurodegeneration and mitochondrial dysfunction

Frantseva, Marina, Perez Velazquez, Jose Luis, Tonkikh, Alexandre, Adamchik, Yana, Carlen, Peter L.·2002·Progress in Brain Research

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Crossref · 10.1016/s0079-6123(02)37015-8

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  • These mechanisms range from inflammatory and glutamate excitotoxicity pathways, which increase metabolic demands of neurons and microglia, to oxidative stress pathways that may impact mitochondrial function.

    “They range from inflammatory and glutamate excitotoxicity pathways (57;58), which increase the metabolic demands of neurons and microglia, to oxidative stress pathways that may impact mitochondrial function (59).”

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Authors
Frantseva, M., Perez Velazquez, J. L., Tonkikh, A., Adamchik, Y., & Carlen, P. L.
Venue
Progress in Brain Research
Publisher
Elsevier
Published
2002
Pages
171-176
ISSN
0079-6123
Cited by
27
References
0

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