The DOI matches its Crossref record; 77 of the 77 references it cites were found in a registry or an index.
Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin
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Citations
Cites
The versatility and universality of calcium signalling
Berridge, M. J., Lipp, P., & Bootman, M. D. (2000) · Nature Reviews Molecular Cell Biology · doi:10.1038/35036035 · Cited by 5,701
ras oncogenes in human cancer: a review.
Bos, J. L. (1989) · PubMed · Cited by 5,061
Marshall, C. J. (1995) · Cell · doi:10.1016/0092-8674(95)90401-8 · Cited by 3,906
Barbacid, M. (1987) · Annual Review of Biochemistry · doi:10.1146/annurev.bi.56.070187.004023 · Cited by 3,885
Mitogen-activated protein kinase pathways
Robinson, M. J., & Cobb, M. H. (1997) · Current Opinion in Cell Biology · doi:10.1016/s0955-0674(97)80061-0 · Cited by 2,474
Calmodulin Plays a Pivotal Role in Cellular Regulation
Cheung, W. Y. (1980) · Science · doi:10.1126/science.6243188 · Cited by 2,438
Clapham, D. E. (1995) · Cell · doi:10.1016/0092-8674(95)90408-5 · Cited by 1,959
Phosphatidylinositol-3-OH kinase direct target of Ras
Rodriguez-Viciana, P., Warne, P. H., Dhand, R., Vanhaesebroeck, B., Gout, I., Fry, M. J., Waterfield, M. D., & Downward, J. (1994) · Nature · doi:10.1038/370527a0 · Cited by 1,954
Proteins regulating Ras and its relatives
Boguski, M. S., & McCormick, F. (1993) · Nature · doi:10.1038/366643a0 · Cited by 1,901
Signal Transduction through MAP Kinase Cascades
(1998) · Advances in Cancer Research · doi:10.1016/s0065-230x(08)60765-4 · Cited by 1,759
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Mechanical stretch triggers rapid epithelial cell division through Piezo1
Gudipaty, S. A., Lindblom, J., Loftus, P. D., Redd, M. J., Edes, K., Davey, C. F., Krishnegowda, V., & Rosenblatt, J. (2017) · Nature · doi:10.1038/nature21407 · Cited by 803
Ca2+ signalling checkpoints in cancer: remodelling Ca2+ for cancer cell proliferation and survival
Roderick, H. L., & Cook, S. J. (2008) · Nature Reviews Cancer · doi:10.1038/nrc2374 · Cited by 731
The many faces of calmodulin in cell proliferation, programmed cell death, autophagy, and cancer
Berchtold, M. W., & Villalobo, A. (2014) · Biochimica et Biophysica Acta (BBA) - Molecular Cell Research · doi:10.1016/j.bbamcr.2013.10.021 · Cited by 320
Mehlmer, N., Wurzinger, B., Stael, S., Hofmann-Rodrigues, D., Csaszar, E., Pfister, B., Bayer, R., & Teige, M. (2010) · The Plant Journal · doi:10.1111/j.1365-313x.2010.04257.x · Cited by 278
Amaral, M. D., & Pozzo-Miller, L. (2007) · The Journal of Neuroscience · doi:10.1523/jneurosci.5499-06.2007 · Cited by 214
Metadata
- Authors
- Agell, N., Bachs, O., Rocamora, N., & Villalonga, P.
- Venue
- Cellular Signalling
- Publisher
- Elsevier BV
- Published
- 2002-08
- Volume
- 14
- Issue
- 8
- Pages
- 649-654
- ISSN
- 0898-6568 (ISSN-L)
- OpenAlex
- W2079437966
- Cited by
- 398
- References
- 77
- FWCI
- 7.56
- Type
- journal article
- Language
- en
- Open access
- closed
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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- CoChat. (2026). Citation check: Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin. https://app.cochat.ai/papers/10.1016/s0898-6568(02)00007-4
- The paper
- Agell, N., Bachs, O., Rocamora, N., & Villalonga, P. (2002). Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin. Cellular Signalling. https://doi.org/10.1016/s0898-6568(02)00007-4
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
