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Cited by 177

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Association of monocyte chemoattractant protein-1 with renal tubular damage in diabetic nephropathy

Morii, Tsukasa, Fujita, Hiroki, Narita, Takuma, Shimotomai, Takashi, Fujishima, Hiromi, Yoshioka, Naomi, Imai, Hirokazu, Kakei, Masafumi, Ito, Seiki·2003·Journal of Diabetes and its Complications
DOI 10.1016/s1056-8727(02)00176-9
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Research works printed but not linked: 24

  1. Banba, 2000, Possible relationship of monocyte chemoattractant protein-1 with diabetic nephropathy, Kidney International, 58, 684, 10.1046/j.1523-1755.2000.00214.x · DOI · Paper page
  2. Boucher, 1986, Characterization of mononuclear subsets in renal cellular interstitial infiltrates, Kidney International, 29, 1043, 10.1038/ki.1986.105 · DOI
  3. Burton, 1996, The role of proteinuria in the progression of chronic renal failure, American Journal of Kidney Diseases, 27, 765, 10.1016/S0272-6386(96)90512-0 · DOI · Paper page
  4. Danoff, 1998, Chemokines in interstitial injury, Kidney International, 53, 1807, 10.1046/j.1523-1755.1998.00920.x · doi:10.1046/j.1523-1755.1998.00920.x
  5. Eddy, 1995, Renal expression of genes that promote interstitial inflammation and fibrosis in rats with protein-overload proteinuria, Kidney International, 47, 1546, 10.1038/ki.1995.218 · DOI · Paper page
  6. Gunder, 1992, Markers for the diagnosis and monitoring of renal tubular lesions, Clinical Nephrology, 38, S3
  7. Howard, 1991, Urinary albumin excretion, transferrin and iron excretion in diabetic patients, Kidney International, 40, 923, 10.1038/ki.1991.295 · DOI
  8. Ito, 2001, Urinary copper excretion in type 2 diabetic patients with nephropathy, Nephron, 88, 307, 10.1159/000046013 · DOI
  9. Kees-Folts, 1991, A lipid chemotactic factor associated with proteinuria and interstitial nephritis induced by protein overload, Journal of the American Society of Nephrology, 2, 548
  10. Kunin, 1978, Enzymuria as a marker of renal injury and disease: studies of N-acetyl-β-glucosaminidase in the general population and in patients with renal disease, Pediatrics, 62, 751, 10.1542/peds.62.5.751 · doi:10.1542/peds.62.5.751
  11. Lloyd, 1997, RANTES and MCP-1 play an important role in the inflammatory phase of crescentic nephritis but only MCP-1 is involved in crescent formation and interstitial fibrosis, Journal of Experimental Medicine, 185, 1371, 10.1084/jem.185.7.1371 · DOI · Paper page
  12. Parving, 1998, Renoprotection in diabetes: genetic and non-genetic risk factors and treatment, Diabetologia, 41, 745, 10.1007/s001250050983 · DOI
  13. Parving, 1983, Diabetic nephropathy and arterial hypertension, Diabetologia, 24, 10, 10.1007/BF00275939 · DOI
  14. Parving, 1988, Prevalence of microalbuminuria, arterial hypertension, retinopathy and neuropathy in patients with insulin dependent diabetes, BMJ [British Medical Journal], 296, 156, 10.1136/bmj.296.6616.156 · DOI · Paper page
  15. Price, 1992, Measurement of N-acetyl-β-glucosaminidase and its isoenzymes in urine methods and clinical applications, European Journal of Clinical Chemistry and Clinical Biochemistry, 30, 693
  16. Prodjosudjadi, 1995, Production and cytokine-mediated regulation of monocyte chemoattractant protein-1 by human proximal tubular epithelial cells, Kidney International, 48, 1477, 10.1038/ki.1995.437 · DOI · Paper page
  17. Remuzzi, 1995, Abnormal protein traffic through the glomerular barrier induces proximal tubular cell dysfunction and causes renal injury, Current Opinion in Nephrology and Hypertension, 4, 339, 10.1097/00041552-199507000-00009 · DOI
  18. Remuzzi, 1999, Nephropathic nature of proteinuria, Current Opinion in Nephrology and Hypertension, 8, 655, 10.1097/00041552-199911000-00001 · DOI
  19. Remuzzi, 1997, Understanding the nature of renal disease progression, Kidney International, 51, 2, 10.1038/ki.1997.2 · DOI · Paper page
  20. Rovin, 1992, Cytokine-induced production of monocyte chemoattractant protein-1 by cultured human mesangial cells, Journal of Immunology, 148, 2148, 10.4049/jimmunol.148.7.2148 · DOI · Paper page
  21. Shore, 1982, Serum and urinary lipoproteins in the human nephrotic syndrome: evidence for renal catabolism of lipoproteins, Metabolism, 31, 258, 10.1016/0026-0495(82)90062-2 · doi:10.1016/0026-0495(82)90062-2
  22. Tarnow, 1994, Prevalence of arterial hypertension in diabetic patients before and after the JNC-V, Diabetes Care, 17, 1247, 10.2337/diacare.17.11.1247 · DOI · Paper page
  23. Wada, 2000, Up-regulation of monocyte chemoattractant protein-1 in tubulointerstitial lesions of human diabetic nephropathy, Kidney International, 58, 1492, 10.1046/j.1523-1755.2000.00311.x · DOI · Paper page
  24. Wang, 1997, Induction of monocyte chemoattractant protein-1 in proximal tubule cells by urinary protein, Journal of the American Society of Nephrology, 8, 1537, 10.1681/ASN.V8101537 · DOI · Paper page

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Metadata

Authors
Morii, T., Fujita, H., Narita, T., Shimotomai, T., Fujishima, H., Yoshioka, N., Imai, H., Kakei, M., & Ito, S.
Venue
Journal of Diabetes and its Complications
Publisher
Elsevier BV
Published
2003-01
Volume
17
Issue
1
Pages
11-15
ISSN
  • 0891-6632 (ISSN-L)
  • 1056-8727
DOI
10.1016/s1056-8727(02)00176-9
OpenAlex
W1974485274
Cited by
177
References
0
FWCI
2.13
Type
journal article
Language
en
Open access
closed
Licence
www.elsevier.com/tdm/userlicense/1.0/ (tdm)

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CoChat. (2026). Citation check: Association of monocyte chemoattractant protein-1 with renal tubular damage in diabetic nephropathy. https://app.cochat.ai/papers/10.1016/s1056-8727(02)00176-9
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Morii, T., Fujita, H., Narita, T., Shimotomai, T., Fujishima, H., Yoshioka, N., Imai, H., Kakei, M., & Ito, S. (2003). Association of monocyte chemoattractant protein-1 with renal tubular damage in diabetic nephropathy. Journal of Diabetes and its Complications. https://doi.org/10.1016/s1056-8727(02)00176-9

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.