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Cited by 344

The DOI matches its Crossref record; 104 of the 104 references it cites were found in a registry or an index.

Neuromodulation of Na+ channels: An unexpected form of cellular platicity

Cantrell, Angela R., Catterall, William A.·2001·Nature Reviews Neuroscience

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Show all 104 references

Research works printed but not linked: 14

  1. Crill, W. E. The effectiveness of distal synaptic inputs on neurons. Ann. NY Acad. Sci. 835, 77–82 (1997). · doi:10.1111/j.1749-6632.1997.tb48619.x
  2. Catterall, W. A. The molecular basis of neuronal excitability. Science 223, 653–661 (1984). · doi:10.1126/science.6320365
  3. Goldin, A. L. et al. Messenger RNA coding for only the α subunit of the rat brain sodium channel is sufficient for expression of functional channels in Xenopus oocytes. Proc. Natl Acad. Sci. USA 83, 7503–7507 (1986). · doi:10.1073/pnas.83.19.7503
  4. Isom, L. L. et al. Functional co-expression of the β1 and type IIA α subunits of sodium channels in a mammalian cell line. J. Biol. Chem. 270, 3306–3312 (1995). · doi:10.1074/jbc.270.7.3306
  5. Murphy, B. J. & Catterall, W. A. Phosphorylation of purified rat brain Na+ channel reconstituted into phospholipid vesicles by protein kinase C. J. Biol. Chem. 267, 16129–16134 (1992). · doi:10.1016/s0021-9258(18)41976-x
  6. Schreibmayer, W., Dascal, N., Lotan, I., Wallner, M. & Weigl, L. Molecular mechanisms of protein kinase C modulation of sodium channel α-subunits expressed in Xenopus oocytes. FEBS Lett. 291, 341–344 (1991). · doi:10.1016/0014-5793(91)81316-z
  7. Godoy, C. M. & Cukierman, S. Diacylglycerol-induced activation of protein kinase C attenuates Na+ currents by enhancing inactivation from the closed state. Pflugers Arch. 429, 245–252 (1994). · doi:10.1007/bf00374319
  8. Cantrell, A. R. et al. Molecular mechanisms underlying convergent regulation of brain Na+ channels by Protein Kinase A and Protein Kinase C. Soc. Neurosci. Abstr. 26, 1908 (2000).
  9. West, J. W., Numann, R., Murphy, B. J., Scheuer, T. & Catterall, W. A. Phosphorylation of a conserved protein kinase C site is required for modulation of Na+ currents in transfected Chinese hamster ovary cells. Biophys. J. 62, 31–33 (1992). · doi:10.1016/s0006-3495(92)81769-1
  10. Maurice, N., Tkatch, T. & Surmeier, J. D1/D5 dopamine receptor activation suppresses rapidly inactivating but not persistent sodium currents in rat prefrontal cortex pyramidal neurons. Soc. Neurosci. Abstr. 26, 1432 (2000).
  11. Alroy, G., Su, H. & Yaari, Y. Protein kinase C mediates muscarinic block of intrinsic bursting in rat hippocampal neurons. J. Physiol. 518, 71–79 (1999). · doi:10.1111/j.1469-7793.1999.0071r.x
  12. Waxman, S. G. & Wood, J. D. Sodium channels: from mechanisms to medicines? Brain Res. Bull. 50, 309–310 (1999). · doi:10.1016/s0361-9230(99)00158-6
  13. Kim, K. J. & Rieke, F. Temporal contrast adaptation in the salamander retina. Invest. Ophthalmol. Vis. Sci. 41 S937 (2000).
  14. Li, Z. et al. Nitric oxide as an autocrine regulator of sodium currents in baroreceptor neurons. Neuron 20, 1039–1049 (1998). · doi:10.1016/s0896-6273(00)80484-5

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Metadata

Authors
Cantrell, A. R., & Catterall, W. A.
Venue
Nature Reviews Neuroscience
Published
2001-06
Volume
2
Issue
6
Pages
397-407
ISSN
  • 1471-003X (ISSN-L)
  • 1471-0048
OpenAlex
W2039072874
Cited by
344
References
104
FWCI
1.00
Type
journal article
Language
en
Open access
closed
Licence
www.springer.com/tdm (tdm)

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CoChat. (2026). Citation check: Neuromodulation of Na+ channels: An unexpected form of cellular platicity. https://app.cochat.ai/papers/10.1038/35077553
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Cantrell, A. R., & Catterall, W. A. (2001). Neuromodulation of Na+ channels: An unexpected form of cellular platicity. Nature Reviews Neuroscience. https://doi.org/10.1038/35077553

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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