Intercellular cannibalism fuels tumor growth
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Internalization in entosis is often but not always followed by demise of internalized cells.
“Often (but not always), internalization is followed by the demise of internalized cells (which are commonly referred to as “entotic cells”) [691–693].”
From Molecular mechanisms of cell death: recommendations of the nomenclature committee on cell death 2018. (2018) · cites this paper as [693]
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Cites
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Cited by
Sorted by citations
Galluzzi, L., Vitale, I., Aaronson, S. A., Abrams, J. M., Adam, D., Agostinis, P., Alnemri, E. S., Altucci, L., Amelio, I., Andrews, D. W., et al. (2018) · Cell Death & Differentiation · doi:10.1038/s41418-017-0012-4 · Cited by 6,680 · see citing works
- Authors
- Pérez, E., & Bergmann, A.
- Affiliations
- Austin College (US)
- University of Massachusetts Chan Medical School (US)
- Corresponding author
- Bergmann, Andreas
- ORCID
- Bergmann, Andreas
- Venue
- Cell Death & Differentiation
- Published
- 2017-03-24
- Volume
- 24
- Issue
- 5
- Pages
- 759-760
- ISSN
- 1350-9047 (ISSN-L)
- 1476-5403
- PMC
- PMC5423114
- OpenAlex
- W2598604780
- Cited by
- 5
- References
- 11
- Citations by year
- 2024: 1 · 2023: 1 · 2019: 1 · 2018: 2
- FWCI
- 0.64
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Licence
- www.springer.com/tdm (tdm)
- Locations
- Cell Death and Differentiation (publishedVersion)
- Europe PMC (PubMed Central) (publishedVersion)
- PubMed Central (publishedVersion)
- PubMed (publishedVersion)
- Funders
- National Institute of General Medical Sciences (R01GM068016, R35GM118330)
- Topics
- Health Sciences › Medicine › Epidemiology › Autophagy in Disease and Therapy
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Cell death mechanisms and regulation
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Cancer Research › Cancer, Hypoxia, and Metabolism
- Keywords
- tumor growth, autophagy, autophagosomes, lysosomal degradation, amino acid recycling, autophagic cell death, tumor cell proliferation, tumor invasion
- MeSH terms
- Animals, Autophagy, Humans, Mice, Transgenic, Models, Biological, Neoplasms, Cell Proliferation
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