Single-cell transcriptomics uncovers distinct molecular signatures of stem cells in chronic myeloid leukemia

Giustacchini, Alice, Thongjuea, Supat, Barkas, Nikolaos, Woll, Petter S, Povinelli, Benjamin J, Booth, Christopher A G, Sopp, Paul, Norfo, Ruggiero +12 more·2017·Nature Medicine
DOI 10.1038/nm.4336

Cited by (423)

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  77. Additional file 5 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  78. Additional file 4 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  79. Additional file 7 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  80. Additional file 2 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  82. Additional file 9 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  84. Additional file 11 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  85. Additional file 3 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML
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  94. Additional file 5 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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  95. Additional file 8 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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  96. Additional file 4 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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  97. Additional file 1 of Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance
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  98. Additional file 3 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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  99. Additional file 1 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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  100. Additional file 7 of Joint analysis of mutational and transcriptional landscapes in human cancer reveals key perturbations during cancer evolution
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