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Cited by 211

The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.

Permanent correction of an inherited ectodermal dysplasia with recombinant EDA

Open access
Gaide, Olivier, Schneider, Pascal·2003·Nature Medicine

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Crossref · 10.1038/nm861

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Citations

Cites

Research works printed but not linked: 20

  1. Pinheiro, M. & Freire-Maia, N. Ectodermal dysplasias: a clinical classification and a causal review. Am. J. Med. Genet. 53, 153–162 (1994). · DOI · Paper page
  2. Monreal, A.W., Zonana, J. & Ferguson, B. Identification of a new splice form of the EDA1 gene permits detection of nearly all X-linked hypohidrotic ectodermal dysplasia mutations. Am. J. Hum. Genet. 63, 380–389 (1998). · DOI · Paper page
  3. Srivastava, A.K. et al. The Tabby phenotype is caused by mutation in a mouse homologue of the EDA gene that reveals novel mouse and human exons and encodes a protein (ectodysplasin-A) with collagenous domains. Proc. Natl. Acad. Sci. USA 94, 13069–13074 (1997). · DOI · Paper page
  4. Yan, M. et al. Two-amino acid molecular switch in an epithelial morphogen that regulates binding to two distinct receptors. Science 290, 523–527 (2000). · DOI · Paper page
  5. Laurikkala, J. et al. Regulation of hair follicle development by the TNF signal ectodysplasin and its receptor Edar. Development 129, 2541–2553 (2002). · DOI · Paper page
  6. Schneider, P. et al. Mutations leading to X-linked hypohidrotic ectodermal dysplasia affect three major functional domains in the tumor necrosis factor family member ectodysplasin-A. J. Biol. Chem. 276, 18819–18827 (2001). · DOI · Paper page
  7. Thesleff, I. & Mikkola, M.L. Death receptor signaling giving life to ectodermal organs. Science STKE 2002, PE22 (2002). · DOI
  8. Srivastava, A.K. et al. Ectodysplasin-A1 is sufficient to rescue both hair growth and sweat glands in Tabby mice. Hum. Mol. Genet. 10, 2973–2981 (2001). · DOI
  9. Rennert, P.D., Browning, J.L. & Hochman, P.S. Selective disruption of lymphotoxin ligands reveals a novel set of mucosal lymph nodes and unique effects on lymph node cellular organization. Int. Immunol. 9, 1627–1639 (1997). · DOI
  10. Colcher, D. et al. Pharmacokinetics and biodistribution of genetically-engineered antibodies. Q. J. Nucl. Med. 42, 225–241 (1998).
  11. Holler, N. et al. Two adjacent trimeric Fas ligands are required for Fas signaling and formation of a death-inducing signaling complex. Mol. Cell. Biol. 23, 1428–1440 (2002). · DOI · Paper page
  12. Kojima, T. et al. TROY, a newly identified member of the tumor necrosis factor receptor superfamily, exhibits a homology with Edar and is expressed in embryonic skin and hair follicles. J. Biol. Chem. 275, 20742–20747 (2000). · DOI
  13. Kristenova-Cermakova, P., Peterka, M., Lisi, S., Lesot, H. & Peterkova, R. Postnatal lower jaw dentition in different phenotypes of tabby mice. Connect. Tissue Res. 43, 283–288 (2002). · DOI
  14. Lesot, H. et al. Mouse molar morphogenesis revisited by three-dimensional reconstruction. II. Spatial distribution of mitoses and apoptosis in cap to bell staged first and second upper molar teeth. Int. J. Dev. Biol. 40, 1017–1031 (1996).
  15. Naito, A. et al. TRAF6-deficient mice display hypohidrotic ectodermal dysplasia. Proc. Natl. Acad. Sci. USA 99, 8766–8771 (2002). · DOI · Paper page
  16. Schmidt-Ullrich, R. et al. Requirement of NF-κB/Rel for the development of hair follicles and other epidermal appendices. Development 128, 3843–3853 (2001). · DOI · Paper page
  17. Doffinger, R. et al. X-linked anhidrotic ectodermal dysplasia with immunodeficiency is caused by impaired NF-κB signaling. Nat. Genet. 27, 277–285 (2001). · DOI · Paper page
  18. Montonen, O. et al. The gene defective in anhidrotic ectodermal dysplasia is expressed in the developing epithelium, neuroectoderm, thymus, and bone. J. Histochem. Cytochem. 46, 281–289 (1998). · DOI
  19. Ersch, J. & Stallmach, T. Assessing gestational age from histology of fetal skin: an autopsy study of 379 fetuses. Obstet. Gynecol. 94, 753–757 (1999).
  20. Schneider, P. Production of recombinant TRAIL and TRAIL receptor: Fc chimeric proteins. Meth. Enzymol. 322, 325–345 (2000). · DOI

Cited by

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Metadata

Authors
Gaide, O., & Schneider, P.
Venue
Nature Medicine
Published
2003-04-07
Volume
9
Issue
5
Pages
614-618
ISSN
  • 1078-8956 (ISSN-L)
  • 1546-170X
OpenAlex
W2058225094
Cited by
211
References
5
FWCI
3.86
Type
journal article
Language
en
Open access
green
Licence
www.springer.com/tdm (tdm)

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