Targeting innate immune mediators in type 1 and type 2 diabetes
Abstract
Type 1 and type 2 diabetes are characterized by chronic inflammation; both diseases involve pancreatic islet inflammation, while systemic low-grade inflammation is a feature of obesity and type 2 diabetes. Long-term activation of the innate immune system impairs insulin secretion and action, and inflammation also contributes to macrovascular and microvascular complications of diabetes. However, despite strong preclinical evidence and proof-of-principle clinical trials demonstrating that targeting inflammatory pathways can prevent cardiovascular disease and other complications in patients with diabetes, there are still no approved treatments for diabetes that target innate immune mediators. Here, we review recent advances in our understanding of the inflammatory pathogenesis of type 1 and type 2 diabetes from a translational angle and point out the critical gaps in knowledge that need to be addressed to guide drug development.
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The pathological microenvironment in diabetic bone defect areas is characterized by hyperglycemia, chronic inflammation, vascular dysfunction, and immune dysregulation.
“The pathological microenvironment in DM bone defect areas is characterized by hyperglycemia, chronic inflammation, vascular dysfunction, and immune dysregulation4–6.”
From Microenvironment-responsive multifunctional enzyme-linked hydrogel for diabetic bone defect regeneration. (2025) · cites this paper as [5]
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References 168 · Cited by 416
- Authors
- Donath, M. Y., Dinarello, C. A., & Mandrup-Poulsen, T.
- Affiliations
- University of Basel (CH)
- University of Colorado Anschutz (US)
- University of Copenhagen (DK)
- Corresponding author
- Donath, Marc Y.
- Venue
- Nature Reviews Immunology
- Published
- 2019-09-09
- Volume
- 19
- Issue
- 12
- Pages
- 734-746
- ISSN
- 1474-1733 (ISSN-L)
- 1474-1741
- OpenAlex
- W2972338204
- Cited by
- 416
- References
- 168
- Citations by year
- 2026: 43 · 2025: 56 · 2024: 83 · 2023: 61 · 2022: 68 · 2021: 51 · 2020: 51 · 2019: 2 · 2017: 1
- FWCI
- 6.27
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Nature reviews. Immunology (publishedVersion)
- PubMed (publishedVersion)
- Research at the University of Copenhagen (University of Copenhagen) (submittedVersion)
- Topics
- Health Sciences › Medicine › Epidemiology › Adipokines, Inflammation, and Metabolic Diseases
- Health Sciences › Medicine › Surgery › Pancreatic function and diabetes
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Diabetes and associated disorders
- Keywords
- type 1 diabetes, type 2 diabetes, innate immunity, inflammation, IL-1β, pancreatic islet inflammation, low-grade inflammation, obesity, insulin secretion, insulin resistance, diabetes complications, anti-inflammatory drug development, inflammatory pathogenesis
- MeSH terms
- Diabetes Mellitus, Type 1, Diabetes Mellitus, Type 2, Glucose, Humans, Immunity, Innate, Insulin Resistance, Interleukin-1, Signal Transduction, Cytokines
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