Cited by 7Redox biology and oxidative stressNeonatal Health and BiochemistryVitamin C and Antioxidants Research

Ascorbate-induced generation of free radical species in normal and glucose-6-phosphate dehydrogenase-deficient erythrocytes

RAKITZIS, EMMANUEL T., PAPANDREOU, PHOTINI T.·1989·Biochemical Society Transactions

Abstract

Conference Article| April 01 1989 Ascorbate-induced generation of free radical species in normal and glucose-6-phosphate dehydrogenase-deficient erythrocytes EMMANUEL T. RAKITZIS; EMMANUEL T. RAKITZIS *Department of Biological Chemistry, University of Athens Medical School, Athens 11527, Greece Search for other works by this author on: This Site PubMed Google Scholar PHOTINI T. PAPANDREOU PHOTINI T. PAPANDREOU †Haematology Laboratory, Pendeli General Children's Hospital, Athens 152 36, Greece Search for other works by this author on: This Site PubMed Google Scholar Biochem Soc Trans (1989) 17 (2): 371–372. https://doi.org/10.1042/bst0170371 Article history Received: September 30 1988 Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Facebook Twitter LinkedIn MailTo Cite Icon Cite Get Permissions Citation EMMANUEL T. RAKITZIS, PHOTINI T. PAPANDREOU; Ascorbate-induced generation of free radical species in normal and glucose-6-phosphate dehydrogenase-deficient erythrocytes. Biochem Soc Trans 1 April 1989; 17 (2): 371–372. doi: https://doi.org/10.1042/bst0170371 Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsBiochemical Society Transactions Search Advanced Search Keywords: G6PD, glucose-6-phosphate dehydrogenase This content is only available as a PDF. © 1989 Biochemical Society1989 Article PDF first page preview Close Modal You do not currently have access to this content.

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Crossref · 10.1042/bst0170371

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  • High-dose intravenous ascorbate caused haemolytic anaemia in patients lacking G6PD.

    “Patients with glucose 6-phosphate dehydrogenase (G6PD) deficiency showed haemolytic anaemia following high-dose intravenous ascorbate therapy56–59.”

    From Targeting cancer vulnerabilities with high-dose vitamin C (2019) · cites this paper as [58]

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Authors
RAKITZIS, E. T., & PAPANDREOU, P. T.
Affiliations
  • National and Kapodistrian University of Athens (GR)
  • Penteli General Children's Hospital (GR)
Venue
Biochemical Society Transactions
Published
1989-04-01
Volume
17
Issue
2
Pages
371-372
ISSN
  • 0300-5127 (ISSN-L)
  • 1470-8752
OpenAlex
W2598451945
Cited by
7
References
0
Citations by year
2022: 1 · 2019: 1
FWCI
0.25
Type
journal article
Language
en
Open access
closed
Locations
Biochemical Society Transactions (publishedVersion)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Redox biology and oxidative stress
  • Health Sciences › Medicine › Pediatrics, Perinatology and Child Health › Neonatal Health and Biochemistry
  • Health Sciences › Nursing › Nutrition and Dietetics › Vitamin C and Antioxidants Research
Keywords
glucose-6-phosphate dehydrogenase deficiency, erythrocytes, vitamin C, free radicals, oxidative stress

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