The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
Adrenergic Receptors in Man: Direct Identification, Physiologic Regulation, and Clinical Alterations
Abstract
THE catecholamines norepinephrine and epinephrine are key regulators of many physiologic events in human beings; norepinephrine acts primarily as a neurotransmitter released from sympathetic-nerve terminals, and epinephrine functions as a circulating hormone released from the adrenal medulla. These catecholamines initiate target-cell responses by binding to specific recognition sites, the adrenergic receptors. That receptors are the initial decoders of extracellular messages is a concept that has guided research on hormone and neurotransmitter action for many years. Studies of adrenergic receptors focused first on physiologic responses that are promoted by adrenergic stimulation and demonstrated the existence of two primary types of adrenergic . . .
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Ginsberg, A. M., Clutter, W. E., Shah, S. D., & Cryer, P. E. (1981) · Journal of Clinical Investigation · doi:10.1172/jci110218 · Cited by 99
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Metadata
- Authors
- Motulsky, H. J., & Insel, P. A.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 1982-07
- Volume
- 307
- Issue
- 1
- Pages
- 18-29
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W1550978342
- Cited by
- 401
- References
- 1
- FWCI
- 16.04
- Type
- journal article
- Language
- en
- Open access
- closed
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- Motulsky, H. J., & Insel, P. A. (1982). Adrenergic Receptors in Man: Direct Identification, Physiologic Regulation, and Clinical Alterations. New England Journal of Medicine. https://doi.org/10.1056/nejm198207013070104
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.