The DOI matches its Crossref record; 24 of the 24 references it cites were found in a registry or an index.
The Role of Complement and Its Receptor in the Elimination of Immune Complexes
Abstract
Information that has accumulated in the past several years has necessitated a radical reexamination of the role of complement in immune-complex disease. The starting point was the recognition in the mid-1970s of an association between a genetic deficiency of complement components and susceptibility to immune-complex disease. More recent work has established that complement itself, by binding to the antigen–antibody complex, can greatly modify the structure and biologic activity of the complex. In addition, the structure and properties of the receptor for the activated third component of complement have been characterized. These new data have led to a reappraisal of the . . .
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Cites
Fearon, D. (1980) · The Journal of Experimental Medicine · doi:10.1084/jem.152.1.20 · Cited by 644
ROSS, S. C., & DENSEN, P. (1984) · Medicine · doi:10.1097/00005792-198409000-00001 · Cited by 550
Kerjaschki, D., & Farquhar, M. (1983) · The Journal of Experimental Medicine · doi:10.1084/jem.157.2.667 · Cited by 501
Primate erythrocyte-immune complex-clearing mechanism.
Cornacoff, J. B., Hebert, L. A., Smead, W. L., VanAman, M. E., Birmingham, D. J., & Waxman, F. J. (1983) · Journal of Clinical Investigation · doi:10.1172/jci110764 · Cited by 489
The Biology and Detection of Immune Complexes
Theofilopoulos, A. N., & Dixon, F. J. (1980) · Advances in Immunology · doi:10.1016/s0065-2776(08)60800-7 · Cited by 467
In situ immune complex formation and glomerular injury
Couser, W. G., & Salant, D. J. (1980) · Kidney International · doi:10.1038/ki.1980.1 · Cited by 379
Iida, K., Mornaghi, R., & Nussenzweig, V (1982) · The Journal of experimental medicine · doi:10.1084/jem.155.5.1427 · Cited by 339
Medof, M. E., Iida, K., Mold, C., & Nussenzweig, V (1982) · The Journal of experimental medicine · doi:10.1084/jem.156.6.1739 · Cited by 334
Membrane Complement Receptors Specific for Bound Fragments of C3
Ross, G. D., & Medof, M. E. (1985) · Advances in Immunology · doi:10.1016/s0065-2776(08)60341-7 · Cited by 308
Ross, G. D., Yount, W. J., Walport, M. J., Winfield, J. B., Parker, C. J., Fuller, C. R., Taylor, R. P., Myones, B. L., & Lachmann, P. J. (1985) · The Journal of Immunology · doi:10.4049/jimmunol.135.3.2005 · Cited by 300
Cited by
The role of complement in inflammation and phagocytosis
Frank, M. M., & Fries, L. F. (1991) · Immunology Today · doi:10.1016/0167-5699(91)90009-i · Cited by 503
Matsumoto, A. K., Kopicky-Burd, J., Carter, R. H., Tuveson, D. A., Tedder, T. F., & Fearon, D. T. (1991) · The Journal of experimental medicine · doi:10.1084/jem.173.1.55 · Cited by 305
Membrane proteins that protect against complement lysis
Morgan, B. P., & Meri, S. (1994) · Springer Seminars in Immunopathology · doi:10.1007/bf01837366 · Cited by 263
Accumulation of immune complexes in glomerular disease is independent of locally synthesized c3
Sheerin, N. S., Abe, K., Risley, P., & Sacks, S. H. (2006) · Journal of the American Society of Nephrology · doi:10.1681/asn.2004070515 · Cited by 22
Metadata
- Authors
- Epstein, F. H., Schifferli, J. A., Ng, Y. C., & Peters, D. K.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 1986-08-21
- Volume
- 315
- Issue
- 8
- Pages
- 488-495
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W2094930358
- Cited by
- 396
- References
- 24
- FWCI
- 8.55
- Type
- journal article
- Language
- en
- Open access
- closed
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- Epstein, F. H., Schifferli, J. A., Ng, Y. C., & Peters, D. K. (1986). The Role of Complement and Its Receptor in the Elimination of Immune Complexes. New England Journal of Medicine. https://doi.org/10.1056/nejm198608213150805
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.