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Cited by 1,269

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Pathophysiology of Progressive Nephropathies

Remuzzi, Giuseppe, Bertani, Tullio·1998·New England Journal of Medicine

Abstract

In patients with renal diseases characterized by proteinuria, the initial insult to the kidney is usually followed by a progressive decline in the glomerular filtration rate. This decline has been thought to be due to changes in renal hemodynamics initiated by the loss of nephrons.1 When renal mass is reduced in rats, the remaining nephrons undergo sudden hypertrophy, with a concomitant lowering of arteriolar resistance and an increase in glomerular plasma flow.2,3 Afferent arteriolar tone decreases more than efferent arteriolar tone, and therefore, the hydraulic pressure in glomerular capillaries rises4 and the amount of filtrate formed by each nephron . . .

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Crossref · 10.1056/nejm199811123392007

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Metadata

Authors
Remuzzi, G., & Bertani, T.
Venue
New England Journal of Medicine
Published
1998-11-12
Volume
339
Issue
20
Pages
1448-1456
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2318993770
Cited by
1,269
References
2
FWCI
43.85
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Pathophysiology of Progressive Nephropathies. https://app.cochat.ai/papers/10.1056/nejm199811123392007
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Remuzzi, G., & Bertani, T. (1998). Pathophysiology of Progressive Nephropathies. New England Journal of Medicine. https://doi.org/10.1056/nejm199811123392007

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.