Cited by 250Signaling Pathways in DiseaseRenal Transplantation Outcomes and TreatmentsImmunotoxicology and immune responses

The immunosuppressive and toxic effects of FK-506 are mechanistically related: pharmacology of a novel antagonist of FK-506 and rapamycin.

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Dumont, F J, Staruch, M J, Koprak, S L, Siekierka, J J, Lin, C S, Harrison, R, Sewell, T, Kindt, V M +2 more·1992·The Journal of experimental medicine

Abstract

FK-506 inhibits Ca(2+)-dependent transcription of lymphokine genes in T cells, and thereby acts as a powerful immunosuppressant. However, its potential therapeutic applications may be seriously limited by several side effects, including nephrotoxicity and neurotoxicity. At present, it is unclear whether these immunosuppressive and toxic effects result from interference with related biochemical processes. FK-506 is known to interact with FK-binding protein-12 (FKBP-12), an abundant cytosolic protein with cis-trans peptidyl-prolyl isomerase activity (PPIase) activity. Because rapamycin (RAP) similarly binds to FKBP-12, although it acts in a manner different from FK-506, by inhibiting T cell responses to lymphokines, such an interaction with FKBP-12 is not sufficient to mediate immunosuppression. Recently, it was found that the complex of FKBP-12 with FK-506, but not with RAP, inhibits the phosphatase activity of calcineurin. Here, we used L-685,818, the C18-hydroxy, C21-ethyl derivative of FK-506, to explore further the role of FKBP-12 in the immunosuppressive and toxic actions of FK-506. Although L-685,818 bound with high affinity to FKBP-12 and inhibited its PPIase activity, it did not suppress T cell activation, and, when complexed with FKBP-12, did not affect calcineurin phosphatase activity. However, L-685,818 was a potent antagonist of the immunosuppressive activity of both FK-506 and RAP. Moreover, L-685,818 did not induce any toxicity in dogs and rats or in a mouse model of acute FK-506 nephrotoxicity, but it blocked the effect of FK-506 in this model. Therefore, FK-506 toxicity involves the disruption of biochemical mechanisms related to those implicated in T cell activation. Like immunosuppression, this toxicity is not due to the inhibition of the PPIase activity of FKBP-12, but may be linked to the inhibition of the phosphatase activity of calcineurin by the drug FKBP-12 complex.

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  • Ascomycin specifically inhibits calcineurin activity after binding to the intracellular immunophilin FKBP-12.

    “Ascomycin (a more water-soluble analog of F K-506, also known in the literature as F K-520 and L -683,590) is an immunosuppressant compound that specifically inhibits calcineurin activity after binding to the intracellular immunophilin FKBP-12 (Dumont et al., 1992).”

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Authors
Dumont, F. J., Staruch, M. J., Koprak, S. L., Siekierka, J. J., Lin, C. S., Harrison, R., Sewell, T., Kindt, V. M., Beattie, T. R., & Wyvratt, M.
Affiliations
Merck & Co., Inc., Rahway, NJ, USA (United States) (US)
Venue
The Journal of experimental medicine
Published
1992-09-01
Volume
176
Issue
3
Pages
751-760
ISSN
  • 0022-1007 (ISSN-L)
  • 1540-9538
OpenAlex
W2169523708
Cited by
250
References
2
Citations by year
2026: 1 · 2025: 1 · 2024: 2 · 2023: 3 · 2022: 1 · 2021: 5 · 2020: 3 · 2019: 2 · 2018: 2 · 2017: 2 · 2016: 2 · 2015: 2 · 2014: 1 · 2013: 4 · 2012: 4
FWCI
7.29
Type
journal article
Language
en
Open access
Locations
  • The Journal of Experimental Medicine (publishedVersion)
  • PubMed Central (publishedVersion, cc-by-nc-sa)
  • PubMed (publishedVersion)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Signaling Pathways in Disease
  • Health Sciences › Medicine › Transplantation › Renal Transplantation Outcomes and Treatments
  • Life Sciences › Immunology and Microbiology › Immunology › Immunotoxicology and immune responses
Keywords
FK506, rapamycin, immunosuppression, nephrotoxicity, neurotoxicity, peptidyl-prolyl isomerase, T cell activation, drug toxicity mechanisms, mTOR signaling, mTOR inhibitors
MeSH terms
Amino Acid Isomerases, Animals, Calmodulin-Binding Proteins, Carrier Proteins, Female, Immunosuppressive Agents, Lymphocyte Activation, Mice, Inbred BALB C, Mice, Inbred C57BL, Phosphoprotein Phosphatases, Polyenes, T-Lymphocytes, Tacrolimus, Peptidylprolyl Isomerase, Calcineurin, Sirolimus, Tacrolimus Binding Proteins, Mice

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