Defective macroautophagic turnover of brain lipids in the TgCRND8 Alzheimer mouse model: prevention by correcting lysosomal proteolytic deficits

Cited by (113)

Items 101–113 of 113

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  8. Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11
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  9. Les APP-CTFs au cœur du processus pathologique de la maladie d’Alzheimer : contribution du système lysosomal-autophagique et de la sécrétion exosomale
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  10. Influence des lipides membranaires sur les interactions protéiques liées aux anomalies endolysosomales dans un modèle neuronal de la maladie d'Alzheimer
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