The DOI matches its Crossref record; 7 of the 7 references it cites were found in a registry or an index.
Genotypes of glutathione transferase M1 and P1 and their significance for lung DNA adduct levels and cancer risk
Open accessAbstract
The A-G polymorphism at codon 104 in the glutathione S-transferase P1 (GSTP1) gene was examined in 138 male lung cancer patients and 297 healthy controls. The patients had significantly higher frequency of the GG genotype (15.9%) and a lower frequency of AA (38.4%) than the controls (9.1% and 51.5%, respectively). The level of hydrophobic DNA-adducts were determined in lung tissue from 70 current smokers. Patients with the GG genotype had a significantly higher adduct level than patients with AA (15.5 +/- 10.2 vs 7.9 +/- 5.1 per 10(8) nucleotides, P = 0.006). We also analyzed the deletion polymorphism in the GSTM1 gene in 135 male patients and 342 controls. The patients were stratified according to histology, smoking dose, age, adduct level and mutational types found in the tumors (Ki-ras and p53 genes). The results consistently indicated that the GSTM1 null genotype was associated with a slightly increased lung cancer risk. When the combined GST M1 and P1 genotypes were examined, patients with the combination null and AG or GG had significantly higher adduct levels than all other genotype combinations (P = 0.011). The distribution of combined genotypes was also significantly different in cases and controls, mainly due to increased frequency of the combination GSTM1 null and GSTP1 AG or GG among patients.
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Cites
Harries, L. (1997) · Carcinogenesis · doi:10.1093/carcin/18.4.641 · Cited by 737
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Glutathione S-transferases and drug resistance
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Different susceptibility to smoking-induced DNA damage among male and female lung cancer patients
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Identification of class-mu glutathione transferase genes GSTM1-GSTM5 on human chromosome 1p13.
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Glutathione S-transferase M1 (GSTM1) deficiency and lung cancer risk.
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"Human Lung Carcinogen- DNA Adduct Levels Mediated by Genetic Polymorphisms In Vivo,"
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Cited by
Watson, M. (1998) · Carcinogenesis · doi:10.1093/carcin/19.2.275 · Cited by 668
Accelerated DNA adduct formation in the lung of the Nrf2 knockout mouse exposed to diesel exhaust
Aoki, Y., Sato, H., Nishimura, N., Takahashi, S., Itoh, K., & Yamamoto, M. (2001) · Toxicology and Applied Pharmacology · doi:10.1006/taap.2001.9176 · Cited by 297
Smoking-related DNA and protein adducts in human tissues
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Progress in understanding the molecular pathogenesis of human lung cancer
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Cancer Risk and Low-Penetrance Susceptibility Genes in Gene-Environment Interactions
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Metadata
- Authors
- Ryberg, D.
- Venue
- Carcinogenesis
- Publisher
- Oxford University Press (OUP)
- Published
- 1997-07-01
- Volume
- 18
- Issue
- 7
- Pages
- 1285-1289
- ISSN
- 0143-3334 (ISSN-L)
- 1460-2180
- OpenAlex
- W2112710602
- Cited by
- 417
- References
- 7
- FWCI
- 7.99
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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