The DOI matches its Crossref record; 26 of the 26 references it cites were found in a registry or an index.
A molecular mechanism for aberrantCFTR-dependent HCO3- transport in cystic fibrosis
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Cites
Pendred syndrome is caused by mutations in a putative sulphate transporter gene (PDS)
Everett, L. A., Glaser, B., Beck, J. C., Idol, J. R., Buchs, A., Heyman, M., Adawi, F., Hazani, E., Nassir, E., Baxevanis, A. D., et al. (1997) · Nature Genetics · doi:10.1038/ng1297-411 · Cited by 1,144
Genetic determination of exocrine pancreatic function in cystic fibrosis.
Kristidis, P., Bozon, D., Corey, M. L., Markiewicz, D., Rommens, J. M., Tsui, L.-C., & Durie, P. R. (1992) · PubMed · Cited by 438
Poulsen, J. H., Fischer, H., Illek, B., & Machen, T. E. (1994) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.91.12.5340 · Cited by 420
Aberrant CFTR-dependent HCO-3 transport in mutations associated with cystic fibrosis
Choi, J. Y., Muallem, D., Kiselyov, K., Lee, M. G., Thomas, P. J., & Muallem, S. (2001) · Nature · doi:10.1038/35065099 · Cited by 412
Physiological Basis of Cystic Fibrosis: A Historical Perspective
QUINTON, P. M. (1999) · Physiological Reviews · doi:10.1152/physrev.1999.79.1.s3 · Cited by 405
Pendrin: an apical Cl−/OH−/HCO3−exchanger in the kidney cortex
Soleimani, M., Greeley, T., Petrovic, S., Wang, Z., Amlal, H., Kopp, P., & Burnham, C. E. (2001) · American Journal of Physiology-Renal Physiology · doi:10.1152/ajprenal.2001.280.2.f356 · Cited by 292
Melvin, J. E., Park, K., Richardson, L., Schultheis, P. J., & Shull, G. E. (1999) · Journal of Biological Chemistry · doi:10.1074/jbc.274.32.22855 · Cited by 275
Knauf, F., Yang, C.-L., Thomson, R. B., Mentone, S. A., Giebisch, G., & Aronson, P. S. (2001) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.141241098 · Cited by 249
Kopelman, H., Corey, M., Gaskin, K., Durie, P., Weizman, Z., & Forstner, G. (1988) · Gastroenterology · doi:10.1016/0016-5085(88)90490-8 · Cited by 244
Identification of an apical Cl−/HCO3−exchanger in the small intestine
Wang, Z., Petrovic, S., Mann, E., & Soleimani, M. (2002) · American Journal of Physiology-Gastrointestinal and Liver Physiology · doi:10.1152/ajpgi.00338.2001 · Cited by 243
Cited by
Sensors and regulators of intracellular pH
Casey, J. R., Grinstein, S., & Orlowski, J. (2010) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm2820 · Cited by 2,218
The SLC26 gene family of multifunctional anion exchangers
Mount, D. B., & Romero, M. F. (2004) · Pfl�gers Archiv European Journal of Physiology · doi:10.1007/s00424-003-1090-3 · Cited by 530
Role of CFTR in epithelial physiology
Saint-Criq, V., & Gray, M. A. (2017) · Cellular and Molecular Life Sciences · doi:10.1007/s00018-016-2391-y · Cited by 433
The SLC26 gene family of anion transporters and channels
Alper, S. L., & Sharma, A. K. (2013) · Molecular Aspects of Medicine · doi:10.1016/j.mam.2012.07.009 · Cited by 408
Molecular Mechanism of Pancreatic and Salivary Gland Fluid and HCO3−Secretion
Lee, M. G., Ohana, E., Park, H. W., Yang, D., & Muallem, S. (2012) · Physiological Reviews · doi:10.1152/physrev.00011.2011 · Cited by 377
Metadata
- Authors
- Ko, S. B. H.
- Venue
- The EMBO Journal
- Published
- 2002-11-01
- Volume
- 21
- Issue
- 21
- Pages
- 5662-5672
- ISSN
- 0261-4189 (ISSN-L)
- 1460-2075
- OpenAlex
- W2169500993
- Cited by
- 329
- References
- 26
- FWCI
- 14.34
- Type
- journal article
- Language
- en
- Open access
- bronze
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- CoChat. (2026). Citation check: A molecular mechanism for aberrantCFTR-dependent HCO3- transport in cystic fibrosis. https://app.cochat.ai/papers/10.1093/emboj/cdf580
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- Ko, S. B. H. (2002). A molecular mechanism for aberrantCFTR-dependent HCO3- transport in cystic fibrosis. The EMBO Journal. https://doi.org/10.1093/emboj/cdf580
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.