Apolipoprotein E: genetic variants provide insights into its structure and function
Abstract
Mahley, R. W.; Innerarity, T. L; Rail, S. C. Jr; Weisgraber, K. H.; Taylor, J. M. Author Information
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Type III hlp can be inherited in either a recessive or a dominant fashion.
“Type III hlp can be inherited in either a recessive or a dominant fashion (3).”
From Preferential association of apolipoprotein E Leiden with very low density lipoproteins of human plasma. (1993) · cites this paper as [3]
Several receptor-binding-defective variants of apoE are associated with impaired catabolism of remnant particles, leading to type III hlp.
“Several receptor-bindingdefective variants of apoE have been identified that are associated with impaired catabolism of remnant particles, leading to the development of type III hyperlipoproteinemia (type III hlp) (3).”
From Preferential association of apolipoprotein E Leiden with very low density lipoproteins of human plasma. (1993) · cites this paper as [3]
There are probably other properties of apoE or additional factors that help determine whether the mode of inheritance of type III hlp will be dominant or recessive.
“Instead, it is probably that there are other properties of apoE or additional factors that help determine whether the mode of inheritance of type III hlp will be dominant or recessive (3, 12, 22).”
From Preferential association of apolipoprotein E Leiden with very low density lipoproteins of human plasma. (1993) · cites this paper as [3]
Cited by
Sorted by citations
Cohn, J. S., Marcoux, C., & Davignon, J. (1999) · Arteriosclerosis, Thrombosis, and Vascular Biology · doi:10.1161/01.atv.19.10.2474 · Cited by 156 · see citing works
de Knijff, P., van den Maagdenberg, A. M., Stalenhoef, A. F., Leuven, J. A., Demacker, P. N., Kuyt, L. P., Frants, R. R., & Havekes, L. M. (1991) · Journal of Clinical Investigation · doi:10.1172/jci115349 · Cited by 100 · see citing works
FEUSSNER, G., FUNKE, H., WENG, W., ASSMANN, G., LACKNER, K. J., & ZIEGLER, R. (1992) · European Journal of Clinical Investigation · doi:10.1111/j.1365-2362.1992.tb01511.x · Cited by 28 · see citing works
Fazio, S., Horie, Y., Weisgraber, K., Havekes, L., & Rall, S. (1993) · Journal of Lipid Research · doi:10.1016/s0022-2275(20)40736-9 · Cited by 23 · see citing works
Self-citationFeussner, G., Scharnagl, H., Scherbaum, C., Acar, J., Dobmeyer, J., Lohrmann, J., Wieland, H., & März, W. (1996) · Journal of Lipid Research · doi:10.1016/s0022-2275(20)39106-9 · Cited by 21 · see citing works
Feussner, G., Albanese, M., & Valencia, A. (1996) · Atherosclerosis · doi:10.1016/0021-9150(96)05870-4 · Cited by 7 · see citing works
- Authors
- Mahley, R. W., Innerarity, T. L., Rail, S. C., Weisgraber, K. H., & Taylor, J. M.
- Affiliations
- University of California, San Francisco (US)
- ORCID
- Mahley, R. W.
- Venue
- Current Opinion in Lipidology
- Published
- 1990-04
- Volume
- 1
- Issue
- 2
- Pages
- 87-95
- ISSN
- 0957-9672 (ISSN-L)
- OpenAlex
- W1530990265
- Cited by
- 88
- References
- 0
- Citations by year
- 2018: 2 · 2015: 1
- FWCI
- 9.68
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Current Opinion in Lipidology (publishedVersion)
- Topics
- Health Sciences › Medicine › Surgery › Lipoproteins and Cardiovascular Health
- Health Sciences › Medicine › Surgery › Cholesterol and Lipid Metabolism
- Health Sciences › Medicine › Physiology › Alzheimer's disease research and treatments
- Keywords
- apolipoprotein E, lipoproteins, lipid metabolism, atherosclerosis
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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