Prevalence of ischemic heart disease among patients with sleep apnea syndrome
Open accessAbstract
Abstract We investigated the prevalence of ischemic heart disease (IHD) in sleep apnea syndrome (SAS) and the presence of coronary risk factors involved in the onset of IHD in 386 subjects with suspected SAS due to heavy snoring. The prevalence of IHD among patients with untreated SAS was found to be 23.8%, and the percentage of patients having SAS complicated with IHD was high among those with moderate or severe SAS. Sleep apnea syndrome patients with IHD also showed high prevalences of hypertension and hyperlipidemia. It appears that sleep apnea aggravates the factors that cause coronary vascular disorders, and is involved in the onset of IHD.
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
Autism, with onset before the age of 3 years, shows global cerebral GM hyperplasia in the first 2 years of life and larger frontal and temporal GM volumes by 4 years, followed by a slower rate of growth in these regions by 7 years.
“For example, autism, with onset before the age of 3 years, shows global cerebral GM hyperplasia in the first 2 years of life (49) and larger frontal and temporal GM volumes by 4 years, followed by a slower rate of growth in these regions by 7 years (50, 51).”
From Dynamic mapping of human cortical development during childhood through early adulthood (2004) · cites this paper as [50]
The Subjects and Methods and Results sections address sleep apnea and ischemic heart disease, not autism or cerebral gray-matter development (page numbers unavailable). Searches for autism, gray-matter hyperplasia, and frontal and temporal growth returned no passage addressing the claim. (LLM, conf=0.78)
Experimental — accuracy not yet measured
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Cites
Association of sleep apnoea with myocardial infarction in men
Hung, J., Whitford, E. G., Hillman, D. R., & Parsons, R. W. (1990) · The Lancet · doi:10.1016/0140-6736(90)91799-g · Cited by 905 · see citing works
Sleep-Disordered Breathing in Men With Coronary Artery Disease
Mooe, T., Rabben, T., Wiklund, U., Franklin, K. A., & Eriksson, P. (1996) · CHEST Journal · doi:10.1378/chest.109.3.659 · Cited by 333 · see citing works
Koehler, U., & Schäfer, H. (1996) · SLEEP · doi:10.1093/sleep/19.4.283 · Cited by 59 · see citing works
Flagged first, then by citations
Research works printed but not linked: 5
- Bonnet M, Carley D, Carskadon M ef al. EEG arousals: Scoring rules and examples. Sleep 1992; 15: 17S184.
- Phillipson EA, Kozar LF. Effect of aging on metabolic respiratory control in sleeping dogs. Am. Rev. Respir. Dis. 1993; 147: 1521-1525.
- Yasuma F, Kozar LF, Kirnoff RJ el a/. Interaction of chemical and mechanical respiratory stimuli in the arousal response to hypoxia. Am. Rev. Respir. Dis. 1991; 143: 1274-1277.
- Noda A, Okada T, Katsumata K, Kayukawa Y, Ohta T. Arousal re- sponse on resumption of breathing between elderly and middle-aged patients with obstructive sleep apnea syndrome. jpn. j . EEC E M C 5. No& A, Okada T, Katsumata K, Yasunla F, Nakashima N, Yokota M. Suppressed cardiac and electroencephalographic arousal on apnea/hy- popnea termination in elderly patients with cerebral idarcti0n.j. Clin. Neurophysiology 1997; 14: 68-72, 1996; 24: 381-391. nation of apnea/hypopnea in aged patients with OSAS. Our findings suggest the arousal response at a termination of apnea/ hypopnea and the patterns of sleep disordered breathing may provide useful information on the pathophysiology of OSAS. REFERENCES
- Maekawa M, Shiomi T. Sleep apnea syndrome and ischemic heart disease. Respiration Res. 1997; 16: 570-574.
Cited by
Sorted by citations
Dynamic mapping of human cortical development during childhood through early adulthood
Gogtay, N., Giedd, J. N., Lusk, L., Hayashi, K. M., Greenstein, D., Vaituzis, A. C., Nugent, T. F., Herman, D. H., Clasen, L. S., Toga, A. W., et al. (2004) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.0402680101 · Cited by 5,604 · see citing works
GABAA Receptor Downregulation in Brains of Subjects with Autism
Fatemi, S. H., Reutiman, T. J., Folsom, T. D., & Thuras, P. D. (2009) · Journal of Autism and Developmental Disorders · doi:10.1007/s10803-008-0646-7 · Cited by 439 · see citing works
- Authors
- MAEKAWA, M., SHIOMI, T., USUI, K., SASANABE, R., & KOBAYASHI, T.
- Affiliations
- Aichi Medical University (JP)
- Corresponding author
- MAEKAWA, MASATO
- Venue
- Psychiatry and Clinical Neurosciences
- Publisher
- Wiley
- Published
- 1998-04
- Volume
- 52
- Issue
- 2
- Pages
- 219-220
- ISSN
- 1323-1316 (ISSN-L)
- 1440-1819
- OpenAlex
- W2024859349
- Cited by
- 31
- References
- 3
- Citations by year
- 2021: 1 · 2020: 1 · 2019: 1 · 2018: 1 · 2016: 3 · 2015: 1
- FWCI
- 0.61
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Locations
- Psychiatry and Clinical Neurosciences (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Physiology › Obstructive Sleep Apnea Research
- Social Sciences › Psychology › Experimental and Cognitive Psychology › Sleep and related disorders
- Health Sciences › Medicine › Pharmacology › Apelin-related biomedical research
- Keywords
- sleep apnea, ischemic heart disease, prevalence, coronary risk factors, hypertension, hyperlipidemia, untreated sleep apnea
- MeSH terms
- Coronary Circulation, Coronary Disease, Cross-Sectional Studies, Female, Humans, Hyperlipidemias, Hypertension, Male, Middle Aged, Risk Factors, Sleep Apnea Syndromes, Snoring, Comorbidity, Incidence, Myocardial Ischemia
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
