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Adipose Tissue: Ability to Respond to Nerve Stimulation in vitro
Abstract
Electrical stimulation of nerves to isolated rat and rabbit adipose tissue in vitro causes production of free fatty acids. Starvation increases the response. The response of white (epididymal) fat is prevented by sympathetic denervation. Direct evidence is provided showing that adipose tissue has the capacity to be an effector organ responsive to the nervous system.
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Cited by
Sympathetic Neuro-adipose Connections Mediate Leptin-Driven Lipolysis
Zeng, W., Pirzgalska, R. M., Pereira, M. M. A., Kubasova, N., Barateiro, A., Seixas, E., Lu, Y.-H., Kozlova, A., Voss, H., Martins, G. G., et al. (2015) · Cell · doi:10.1016/j.cell.2015.08.055 · Cited by 490
Sympathetic and sensory innervation of brown adipose tissue
Bartness, T. J., Vaughan, C. H., & Song, C. K. (2010) · International Journal of Obesity · doi:10.1038/ijo.2010.182 · Cited by 459
Influence of Perivascular Adipose Tissue on Rat Aortic Smooth Muscle Responsiveness
Soltis, E. E., & Cassis, L. A. (1991) · Clinical and Experimental Hypertension. Part A: Theory and Practice · doi:10.3109/10641969109042063 · Cited by 382
Bergström, S. (1967) · Science · doi:10.1126/science.157.3787.382 · Cited by 342
Sensory and sympathetic nervous system control of white adipose tissue lipolysis
Bartness, T. J., Shrestha, Y. B., Vaughan, C. H., Schwartz, G. J., & Song, C. K. (2010) · Molecular and Cellular Endocrinology · doi:10.1016/j.mce.2009.08.031 · Cited by 289
Metadata
- Authors
- Correll, J. W.
- Venue
- Science
- Published
- 1963-04-26
- Volume
- 140
- Issue
- 3565
- Pages
- 387-388
- ISSN
- 0036-8075 (ISSN-L)
- 1095-9203
- OpenAlex
- W2027547386
- Cited by
- 212
- References
- 0
- FWCI
- 55.66
- Type
- journal article
- Language
- en
- Open access
- closed
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
