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Cited by 317

The DOI matches its Crossref record; 21 of the 21 references it cites were found in a registry or an index.

Baroreceptor Activation Reduces Reactivity to Noxious Stimulation: Implications for Hypertension

Dworkin, B. R., Filewich, R. J., Miller, N. E., Craigmyle, N., Pickering, T. G.·1979·Science
DOI 10.1126/science.472749
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Abstract

The hypothesis was tested that an acute rise of blood pressure may reduce reactivity to noxious stimuli through a baroreceptor-mediated reduction of cerebral arousal. When blood pressure was raised by an infusion of phenylephrine, rats showed less running to terminate or avoid noxious stimuli than during saline infusions. This effect was not seen in rats with denervated baroreceptors. The results suggest that a rise of blood pressure could have motivational consequences significant for human hypertension.

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Crossref · 10.1126/science.472749

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Metadata

Authors
Dworkin, B. R., Filewich, R. J., Miller, N. E., Craigmyle, N., & Pickering, T. G.
Venue
Science
Publisher
American Association for the Advancement of Science (AAAS)
Published
1979-09-21
Volume
205
Issue
4412
Pages
1299-1301
ISSN
  • 0036-8075 (ISSN-L)
  • 1095-9203
DOI
10.1126/science.472749
OpenAlex
W2065823549
Cited by
317
References
21
FWCI
15.58
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Baroreceptor Activation Reduces Reactivity to Noxious Stimulation: Implications for Hypertension. https://app.cochat.ai/papers/10.1126/science.472749
The paper
Dworkin, B. R., Filewich, R. J., Miller, N. E., Craigmyle, N., & Pickering, T. G. (1979). Baroreceptor Activation Reduces Reactivity to Noxious Stimulation: Implications for Hypertension. Science. https://doi.org/10.1126/science.472749

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.