The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
Recurrent GNAS Mutations Define an Unexpected Pathway for Pancreatic Cyst Development
Open accessAbstract
A mutation in the gene GNAS serves as a marker for pancreatic cysts that can progress to become invasive adenocarcinomas, guiding therapy.
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Citations
Cites
Cystic precursors to invasive pancreatic cancer
Matthaei, H., Schulick, R. D., Hruban, R. H., & Maitra, A. (2011) · Nature Reviews Gastroenterology & Hepatology · doi:10.1038/nrgastro.2011.2 · Cited by 193
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Cited by
Detection of Circulating Tumor DNA in Early- and Late-Stage Human Malignancies
Bettegowda, C., Sausen, M., Leary, R. J., Kinde, I., Wang, Y., Agrawal, N., Bartlett, B. R., Wang, H., Luber, B., Alani, R. M., et al. (2014) · Science Translational Medicine · doi:10.1126/scitranslmed.3007094 · Cited by 5,067
Self-citationInternational consensus guidelines 2012 for the management of IPMN and MCN of the pancreas
Tanaka, M., Fernández-del Castillo, C., Adsay, V., Chari, S., Falconi, M., Jang, J.-Y., Kimura, W., Levy, P., Pitman, M. B., Schmidt, C. M., et al. (2012) · Pancreatology · doi:10.1016/j.pan.2012.04.004 · Cited by 2,354
Self-citationRyan, D. P., Hong, T. S., & Bardeesy, N. (2014) · New England Journal of Medicine · doi:10.1056/nejmra1404198 · Cited by 2,348
Kamisawa, T., Wood, L. D., Itoi, T., & Takaori, K. (2016) · The Lancet · doi:10.1016/s0140-6736(16)00141-0 · Cited by 2,212
Self-citationIntegrated genomic characterization of pancreatic ductal adenocarcinoma
Raphael, B. J., Hruban, R. H., Aguirre, A. J., Moffitt, R. A., Yeh, J. J., Stewart, C., Robertson, A. G., Cherniack, A. D., Gupta, M., Getz, G., et al. (2017) · Cancer Cell · doi:10.1016/j.ccell.2017.07.007 · Cited by 2,019
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Metadata
- Authors
- Wu, J., Matthaei, H., Maitra, A., Dal Molin, M., Wood, L. D., Eshleman, J. R., Goggins, M., Canto, M. I., Schulick, R. D., Edil, B. H., Wolfgang, C. L., Klein, A. P., Diaz, L. A., Allen, P. J., Schmidt, C. M., Kinzler, K. W., Papadopoulos, N., Hruban, R. H., & Vogelstein, B.
- Venue
- Science Translational Medicine
- Published
- 2011-07-20
- Volume
- 3
- Issue
- 92
- Pages
- 92ra66-92ra66
- ISSN
- 1946-6234 (ISSN-L)
- 1946-6242
- OpenAlex
- W1975940378
- Cited by
- 782
- References
- 1
- FWCI
- 27.02
- Type
- journal article
- Language
- en
- Open access
- green
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- CoChat. (2026). Citation check: Recurrent GNAS Mutations Define an Unexpected Pathway for Pancreatic Cyst Development. https://app.cochat.ai/papers/10.1126/scitranslmed.3002543
- The paper
- Wu, J., Matthaei, H., Maitra, A., Dal Molin, M., Wood, L. D., Eshleman, J. R., Goggins, M., Canto, M. I., Schulick, R. D., Edil, B. H., Wolfgang, C. L., Klein, A. P., Diaz, L. A., Allen, P. J., Schmidt, C. M., Kinzler, K. W., Papadopoulos, N., Hruban, R. H., & Vogelstein, B. (2011). Recurrent GNAS Mutations Define an Unexpected Pathway for Pancreatic Cyst Development. Science Translational Medicine. https://doi.org/10.1126/scitranslmed.3002543
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
