The DOI matches its Crossref record; 46 of the 47 references it cites were found in a registry or an index.
Dual suppression of estrogenic and inflammatory activities for targeting of endometriosis
Open accessAbstract
The estrogen-dependent inflammatory and neuroangiogenic activities that drive endometriosis can be suppressed by estrogen receptor ligands in mice.
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“ESR2 ligands acting as estrogen antagonists in endometriotic tissue show promising results in murine models and should also be considered as potential therapeutics (Zhao et al., 2015).”
From Endometriosis and nuclear receptors (2019) · cites this paper as Zhao et al., 2015
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Cites
Giudice, L. C., & Kao, L. C. (2004) · The Lancet · doi:10.1016/s0140-6736(04)17403-5 · Cited by 3,166
Bulun, S. E. (2009) · New England Journal of Medicine · doi:10.1056/nejmra0804690 · Cited by 1,772
Dupont, S., Krust, A., Gansmuller, A., Dierich, A., Chambon, P., & Mark, M. (2000) · Development · doi:10.1242/dev.127.19.4277 · Cited by 1,064
McLaren, J. F., Prentice, A., Jones, D. S., Millican, S. A., Müller, K. H., Sharkey, A., & Smith, S. K. (1996) · Journal of Clinical Investigation · doi:10.1172/jci118815 · Cited by 499
Bacci, M., Capobianco, A., Monno, A., Cottone, L., Di Puppo, F., Camisa, B., Mariani, M., Brignole, C., Ponzoni, M., Ferrari, S., et al. (2009) · The American Journal of Pathology · doi:10.2353/ajpath.2009.081011 · Cited by 458
Berkley, K. J., Rapkin, A. J., & Papka, R. E. (2005) · Science · doi:10.1126/science.1111445 · Cited by 455
Role of Estrogen Receptor-β in Endometriosis
Bulun, S., Monsavais, D., Pavone, M., Dyson, M., Xue, Q., Attar, E., Tokunaga, H., & Su, E. (2012) · Seminars in Reproductive Medicine · doi:10.1055/s-0031-1299596 · Cited by 321
Anaf, V., Simon, P., El Nakadi, I., Fayt, I., Simonart, T., Buxant, F., & Noel, J.-C. (2002) · Human Reproduction · doi:10.1093/humrep/17.7.1895 · Cited by 317
An ADIOL-ERβ-CtBP Transrepression Pathway Negatively Regulates Microglia-Mediated Inflammation
Saijo, K., Collier, J. G., Li, A. C., Katzenellenbogen, J. A., & Glass, C. K. (2011) · Cell · doi:10.1016/j.cell.2011.03.050 · Cited by 295
Self-citationEndometriosis: hormone regulation and clinical consequences of chemotaxis and apoptosis
dos Reis, F. M., Petraglia, F., & Taylor, R. N. (2013) · Human Reproduction Update · doi:10.1093/humupd/dmt010 · Cited by 273
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Research works printed but not linked: 1
- 10.1096/fj.10.163428 · DOI
Cited by
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Endometriosis: Etiology, pathobiology, and therapeutic prospects
Saunders, P. T. K., & Horne, A. W. (2021) · Cell · doi:10.1016/j.cell.2021.04.041 · Cited by 1,090
The Immunopathophysiology of Endometriosis
Symons, L. K., Miller, J. E., Kay, V. R., Marks, R. M., Liblik, K., Koti, M., & Tayade, C. (2018) · Trends in Molecular Medicine · doi:10.1016/j.molmed.2018.07.004 · Cited by 601
The Pathogenesis of Endometriosis: Molecular and Cell Biology Insights
Laganà, A. S., Garzon, S., Götte, M., Viganò, P., Franchi, M. P., Ghezzi, F., & Martin, D. C. (2019) · International Journal of Molecular Sciences · doi:10.3390/ijms20225615 · Cited by 503
Han, S. J., Jung, S. Y., Wu, S.-P., Hawkins, S. M., Park, M. J., Kyo, S., Qin, J., Lydon, J. P., Tsai, S. Y., Tsai, M.-J., et al. (2015) · Cell · doi:10.1016/j.cell.2015.10.034 · Cited by 380
Role of estrogen receptors in health and disease
Chen, P., Li, B., & Ou-Yang, L. (2022) · Frontiers in Endocrinology · doi:10.3389/fendo.2022.839005 · Cited by 339
Metadata
- Authors
- Zhao, Y., Gong, P., Chen, Y., Nwachukwu, J. C., Srinivasan, S., Ko, C., Bagchi, M. K., Taylor, R. N., Korach, K. S., Nettles, K. W., Katzenellenbogen, J. A., & Katzenellenbogen, B. S.
- Affiliations
- University of Illinois Urbana-Champaign (US)
- Scripps Research Institute (US)
- Wake Forest University Health Sciences (US)
- National Institute of Environmental Health Sciences (US)
- Corresponding author
- Katzenellenbogen, Benita S.
- ORCID
- Venue
- Science Translational Medicine
- Published
- 2015-01-21
- Volume
- 7
- Issue
- 271
- Pages
- 271ra9-271ra9
- ISSN
- 1946-6234 (ISSN-L)
- 1946-6242
- PMC
- PMC4790140
- OpenAlex
- W2152982261
- Cited by
- 170
- References
- 47
- Citations by year
- 2026: 10 · 2025: 16 · 2024: 18 · 2023: 11 · 2022: 14 · 2021: 9 · 2020: 15 · 2019: 17 · 2018: 18 · 2017: 12 · 2016: 19 · 2015: 11
- FWCI
- 22.82
- Type
- journal article
- Language
- en
- Open access
- green
- www.ncbi.nlm.nih.gov
- Locations
- Science Translational Medicine (publishedVersion)
- PubMed (publishedVersion)
- PubMed Central (acceptedVersion)
- Funders
- NIH (U54 HD055787, PHS 5R01DK015556, 5R01DK077085)
- Frenchman's Creek Women for Cancer Research
- Topics
- Health Sciences › Medicine › Reproductive Medicine › Endometriosis Research and Treatment
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Endocrinology › Estrogen and related hormone effects
- Physical Sciences › Environmental Science › Health, Toxicology and Mutagenesis › Effects and risks of endocrine disrupting chemicals
- Keywords
- endometriosis, estrogen receptor ligands, estrogen receptor alpha, estrogen receptor beta, anti-inflammatory activity, angiogenesis, neurogenesis, endometriotic stromal cells, fertility preservation
- MeSH terms
- Letrozole, Animals, Anti-Inflammatory Agents, Cell Survival, Cells, Cultured, Disease Models, Animal, Drug Therapy, Combination, Endometriosis, Estrogens, Female, Fertility, Humans, Indazoles, Inflammation, Ligands, Macrophages, Mice, Inbred C57BL, Mice, Transgenic, Neurons, Nitriles, Receptors, Estrogen, Triazoles, Treatment Outcome, Stromal Cells, Disease Progression, Embryonic Stem Cells
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