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Quinolone resistance-determining region in the DNA gyrase gyrB gene of Escherichia coli
Open accessAbstract
Thirteen spontaneous quinolone-resistant gyrB mutants of Escherichia coli KL16, including two that were examined previously, were divided into two types according to their quinolone resistance patterns. Type 1 mutants were resistant to all the quinolones tested, while type 2 mutants were resistant to acidic quinolones and were hypersusceptible to amphoteric quinolones. Nucleotide sequence analysis disclosed that all nine type 1 mutants had a point mutation from aspartic acid to asparagine at amino acid 426 and that all four type 2 mutants had a point mutation from lysine to glutamic acid at amino acid 447.
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Cited by
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Antibiotic Resistance in Bacteria—A Review
Urban-Chmiel, R., Marek, A., Stępień-Pyśniak, D., Wieczorek, K., Dec, M., Nowaczek, A., & Osek, J. (2022) · Antibiotics · doi:10.3390/antibiotics11081079 · Cited by 580
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Pirnay, J.-P., Djebara, S., Steurs, G., Griselain, J., Cochez, C., De Soir, S., Glonti, T., Spiessens, A., Vanden Berghe, E., Green, S., et al. (2024) · Nature Microbiology · doi:10.1038/s41564-024-01705-x · Cited by 474
Plasmid-mediated quinolone resistance: an update
Rodríguez-Martínez, J. M., Velasco, C., Pascual, Á., Cano, M. E., Martínez-Martínez, L., Martínez-Martínez, L., & Pascual, Á. (2011) · Journal of Infection and Chemotherapy · doi:10.1007/s10156-010-0120-2 · Cited by 306
Metadata
- Authors
- Yoshida, H., Bogaki, M., Nakamura, M., Yamanaka, L. M., & Nakamura, S.
- Venue
- Antimicrobial Agents and Chemotherapy
- Publisher
- American Society for Microbiology
- Published
- 1991-08
- Volume
- 35
- Issue
- 8
- Pages
- 1647-1650
- ISSN
- 0066-4804 (ISSN-L)
- 1098-6596
- OpenAlex
- W2154806021
- Cited by
- 777
- References
- 0
- FWCI
- 20.03
- Type
- journal article
- Language
- en
- Open access
- green
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- Yoshida, H., Bogaki, M., Nakamura, M., Yamanaka, L. M., & Nakamura, S. (1991). Quinolone resistance-determining region in the DNA gyrase gyrB gene of Escherichia coli. Antimicrobial Agents and Chemotherapy. https://doi.org/10.1128/aac.35.8.1647
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