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Cited by 32Neonatal Respiratory Health ResearchNeutrophil, Myeloperoxidase and Oxidative MechanismsRespiratory Support and Mechanisms

The DOI matches its Crossref record; 68 of the 68 references it cites were found in a registry or an index.

Antioxidants in neonatal lung disease.

Open access
Fardy, C. H., Silverman, M.·1995·Archives of Disease in Childhood - Fetal and Neonatal Edition
DOI 10.1136/fn.73.2.f112PMC PMC2528507
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Abstract

During Ag stimulation of T cells, the recognition of B7 molecules by the CD28 costimulatory receptor increases the level of c-Fos, a component of the AP-1 transactivator known to bind the 5′ Il2 gene enhancer. In this study, we show that the costimulation of Fos transcription by CD28 is associated with increased binding of p300/CREB-binding protein (CBP) molecules at the Fos promoter, and is blocked by an adenoviral E1A molecular antagonist of p300/CBP. Furthermore, transcriptional activation by a C-terminal domain of CBP is strengthened when CD28 molecules are actively signaling. This increased amount and activity of p300/CBP molecules at the Fos gene correlated with higher histone H4 acetylation and RNA polymerase II association with the promoter. These data suggest a global mechanism whereby CD28 signaling influences the rate and intensity of new gene expression during Ag recognition via direct control over the coactivator function of p300/CBP.

Overview

Integrity signals

Registry

Crossref · 10.1136/fn.73.2.f112

Cites

68 of 68 references verified0 citation errors · 0 not checkable

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Verification

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Citations

Cites

Research works printed but not linked: 3

  1. Raffin TA, Simon LM, Braun D. Impairment of phagocytosis by moderate hypoxia (40-60% oxygen) in lung macrophages. Lab Invest 1980; 42: 622.
  2. Kooy NW, Royall JA, Ye YZ, Kelly DP, Beckman JS. Peroxynitrite is produced during human adult respiratory distress syndrome. Am Jf Respir Crit Care Med 1994; 149: A814.
  3. Guitierrez H, Pitt B, Brockens M, Chumley P, Freeman B. Endogenously-produced and exogenously-administered nitric oxide modulates superoxide-dependant lung injury. Am Jf Respir Crit Care Med 1944; 149: A550.

Cited by

See all 32

Metadata

Authors
Fardy, C. H., & Silverman, M.
Affiliations
Leicester Royal Infirmary (GB)
ORCID
Silverman, M.
Venue
Archives of Disease in Childhood - Fetal and Neonatal Edition
Publisher
BMJ
Published
1995-09
Volume
73
Issue
2
Pages
F112-F117
ISSN
  • 1359-2998 (ISSN-L)
  • 1468-2052
DOI
10.1136/fn.73.2.f112
PMC
PMC2528507
OpenAlex
W2023454634
Cited by
32
References
68
Citations by year
2024: 1 · 2023: 1 · 2019: 2 · 2014: 1 · 2013: 1 · 2012: 1
FWCI
0.97
Type
journal article
Language
en
Open access
Locations
  • Archives of Disease in Childhood Fetal & Neonatal (publishedVersion)
  • PubMed Central (publishedVersion)
  • PubMed (publishedVersion)
Topics
  • Health Sciences › Medicine › Pulmonary and Respiratory Medicine › Neonatal Respiratory Health Research
  • Life Sciences › Immunology and Microbiology › Immunology › Neutrophil, Myeloperoxidase and Oxidative Mechanisms
  • Health Sciences › Medicine › Pulmonary and Respiratory Medicine › Respiratory Support and Mechanisms
Keywords
antioxidants, neonatal lung disease, CD28 costimulation, AP-1, adenoviral E1A, histone H4 acetylation, RNA polymerase II, T cell activation
MeSH terms
Antioxidants, Chronic Disease, Free Radicals, Humans, Infant, Newborn, Infant, Premature, Lung Diseases, Reactive Oxygen Species

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Data sources

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.